0:01
SNI Digital Innovations in Learning.
0:05
In association with the Medical News Network
0:11
are pleased to present another in the SNI Digital Investigative Series on achieving health longevity
0:23
in a talk given by Russell Blalock on the new cause of Parkinson's disease Alzheimer's dementia and ALS what you need to know. Russell Blalock is the
0:40
head of theoretical neuroscience research and the associate editor-in-chief of the neuroinflammation section of surgical neurology international He's also a certified clinical nutritionist. He's the
0:57
creator and editor of the Blalock Wellness Report. and the author of multiple books, scientific papers, and a health commentator on radio TV and the epic times.
1:13
These are some of the books that Dr. Blalock has published. They're available on amazoncom.
1:20
Natural Solutions for Liver Problems, the Liver Care is the title, Natural Strategies for Cancerous Patients, Dr. Blalock's Prescriptions for Natural Health, and a subject of this talk, a book
1:36
on excitotoxins,
1:39
and also a book with Dr. Osmond on the China virus, What is the Truth?
1:46
He's published numerous papers in multiple journals, and notably in SI, he's published papers that are leading publications on COVID Call an update, watch the truth. the dangers of COVID-19
2:02
injections
2:05
and other papers on degenerative disease and Parkinson's disease,
2:11
papers on viruses in the tumor microcell environment, and associated cancer aggressiveness by virus on co-modulation.
2:25
Key to this talk are a series of papers that we will repeat at the end with the references so you can prepare to take screenshots of them for references. On one, the biochemical basis of
2:39
neurodegenerative disease, the role of immuno-excitotoxicity
2:46
in ways to attenuate it,
2:49
the reference is given at the bottom.
2:52
Parkinson's disease, microglial, macrophage-induced communal, excited, or toxicity. As the central mechanism of neurodegeneration, again with the references in scientific surgical neurology
3:07
international,
3:10
additive aluminum as a cause of induced immunoexcitertoxicity
3:16
resulting in neurodevelopmental and neurodegenerative disorders.
3:23
And immunoexcitertoxicity is
3:26
a central mechanism of the idiopathology and treatment of autism spectrum disorders.
3:37
So the major papers which are available in surgical neurology international, which you'll be able to screenshot at the end and look up, contain all the references for the subject matter that he's
3:50
going to present, obviously looks like he's being tied together with the knowledge of immuno-excited toxicity.
4:01
Dr. Blalock also has a monthly nutritional newsletter, which he's published for 20 years. If you're interested, you can subscribe to The Wellness Report at Newsmaxcom.
4:17
Russell, let me show you this video that just came from about a day ago after the presidential debate.
4:25
Able to write what about his debt, we'd be able to help make sure that all those things we need to do, child care, healthcare, making sure that we continue to strengthen our healthcare system,
4:35
making sure that we're able to make every single solitary person eligible for what I've been able to do with the
4:43
COVID, excuse me, with dealing with everything we have to do with,
4:50
what,
4:52
if we finally beat Medicare? Thank you, President Biden, President Trump was right. He did beat Medicaid, beat it to death. It's an example of the president and we've seen him over now since he
5:06
really took office. He had signs and symptoms of what we talked about that. And it's now progressed to this point where you saw the stiff-like faces, fumbling, couldn't remember things. Obviously,
5:22
he has a problem with cognitive
5:28
issues, problem remembering And he walks, you see the stiff and he falls. These are talked to a neurologist friend that you know a minor who says that he has a form of dementia, Alzheimer's
5:41
dementia is his Parkinson's, actually dementia and Lewybody dementia.
5:47
It's confusing for the audience, but these are forms of dementia that are serious and progressive. And it leads into something that you've talked about for many years and you've worked on, What are
6:02
the cause of these neurodegenerative diseases of the nervous system? 'Cause if I'm the average citizen, I go on the internet and I look, I went on to the various different websites and they said,
6:15
well, nobody knows what the causes are. There's a small percentage that are genetic. And then they talk about toxic substances. And I think we should start from there because you've had
6:28
observations from the time you were a resident on about these diseases and tell us about what you see and what the differences are and how does aluminum figure into all this? So I'm giving you a lot
6:42
to do there, but can you tackle this here and looking at what we just saw?
6:49
I think so, you know, my father died of Parkinson's. So I know what it looks like. I know what he went through.
6:60
persisted for years before he finally deteriorated to the point where he died, and we see this in other cases, you know, when I was a resident, I spent over six months on neurology and ran it by
7:13
myself, just the attending and me, because all the neurology residents left
7:21
So I got to treat all of this as a neurologist, and what I observed early on is I began to look at these agents, and this is back during the time, about 20 years ago, they discovered that all
7:39
cases seem to be somehow related to exposure to environmental toxin
7:47
They picked out pesticides, herbicides, and fungicides. Manab was the number one one, along with some of the organo. chlorines or dental phosphates. And so I began to look at these other things.
8:04
We had metals, we had lead, we had aluminum, we had mercury. And so I said, well, there's got to be a common denominator. They're all doing something. And so I looked up each one of them.
8:17
Each one of them was a microglial activator.
8:22
And if you looked in the pathology of this disease, the microglial was the cause of the degeneration, not the chemical itself directly. Let me just ask, can we interrupt our second because we may
8:35
have some people who are not medically knowledgeable. What is the microglia? Are those some protective cells in the nervous system? Tell us about that. Well, the microglia is the brain's immune
8:49
cell. Okay. Okay, they're also in the spinal cord And they're primarily designed as a immune cell. And we have them from birth, and they gradually develop. Like immune cells, they can move
9:04
around in the brain. Several centimeters. And we know that most microglia are sleep all the time, what they call resting microglia. But it's not really resting. What it's doing, it's sending out
9:20
little extensions, which we call pseudopodia, and they're analyzing the spinal fluid. It's floating around the brain cell. To make sure it doesn't have too high glutamate level, glutamate is an
9:37
amino acid, and it's an excitotoxin. And so it's constantly testing it, because if the level is too high, it'll interfere with brain function. You start getting a lot static If it gets higher
9:51
than that, it begins to kill brain cell.
9:56
Most of these diseases we call neurodegenerative diseases are because of excess
10:03
glutamate and overactivity of the microglia. And what happens is when you stimulate this microglia, it becomes active and it changes from this resting state, which can actually help repair the
10:16
nervousers. It changes into an active microglia. When it's activated, it switches over and it starts secreting high levels of glutamate and can destroy things, including connections and nerve
10:33
cells, brain cells, and spinal cord cells. And so if we look at all these diseases, they all show evidence that the damage was done by excitotoxicity.
10:46
And that the immune reaction was not severe enough to account for what they were seeing pathologically and all of these neuroducine and diseases. I wrote an article for your journal called the
10:58
biochemical basis of immuno-excitotoxicity, which explains it in some detail.
11:06
But we begin to look because of this mechanism as what chemicals would do that and what chemicals would reverse that or stop them. And we know that there's certain natural compounds that turn off the
11:22
microglia, they go back to resting and they start repairing the nervous system. And we know that other chemicals in our environment actually turn on these microglia and they become destructive
11:36
elements. And so when they looked in the island of Guam, they were amazed that these people
11:46
would develop Parkinson's disease and then they'd go into Alzheimer's disease. And one of the first diseases they developed
11:57
myotropic lateral sclerosis, which is the destruction of the motor nerve, the anterior motor cell inside the spinal cord, which makes you move.
12:07
So it was moving, it was progressing, it was changing. It went from the spinal cord to the brain, the brain stem, to the part of the brain that has to do with memory and learning, the
12:19
hippocampus, and the part of the brain that has to do with Parkinson's disease,
12:35
which is substantia nigra in the striatum. So they looked at these areas and they said, well, the highest microglial population in the brain is in the substantia nigra. The second highest is in
12:40
the hippocampus. These are the two areas most affected. And then they noticed, well, if you look at the olfactory nerves, the microglia and the olfactory nerves And the olfactory bulb, which is.
12:58
closest to the brain, is also activated. And it is also producing this change. And these microglia entered the brain through what's called an interrinal cortex, which is where the olfactory nerve
13:12
goes into the brain. And then it spreads to the front lobe, temporal lobe, and the pryolob, three most affected areas of the brain in Alzheimer's disease
13:26
And so they found there was two areas in which there seemed to be a progression coming from the spinal cord or the air, you breathe. One was the vagus nerve, and the other was olfactory nerve and
13:41
the crayon nerves, especially trigeminal nerves, lower crayon nerve. And so they looked at the microglia in these areas. They were very active, and they were moving
13:55
So even though he started there, And then they say, Well, where is this coming from? Well, they found that it was coming from the GI tract and the air you breathe. And in the GI tract, if you
14:09
look at it, for instance, the Parkinson's disease, you see alpha-synuclein in the nerves, in the wall of the colon and intestine. And it seems to be progressing towards the brain
14:25
What was not known at the time was, well, it was not appreciated, it was known, was an aluminum is a very inflammatory comable.
14:37
Mercury can do that, so can let. But aluminum was the worst. It seemed to be the thing that was traveling from the GI tract to the brain. You're inhaling a small molecule of aluminum coming
14:53
through the nasal tract and it was in the brain. Well, then they said, Well, let's look at the aluminum level in these diseases. So they looked at it in ALS. The aluminum level in the spine and
15:07
the nerves that were affected was sky high. They looked in the brain and said, Well, we know Alzheimer's starts in the entorhinal cortex and goes to these other areas. Which area of the brain has
15:20
the highest aluminum? Well,
15:25
it was the olfactory track An olfactory bulb in the entorhinal cortex, where it was entering from the air, from the atmosphere.
15:33
And so they begin to see, well, there's a process here. Something is moving, this toxic metal is moving two places, one from the geotract, and one from the air.
15:47
And it seemed to fit everything we see. So you look at a pathophysiologically, it answers a lot of questions, Everybody just can't scratch their head and didn't know why.
15:60
And this idea that, Oh, this is an idiopathic disease. We don't know what causes it. Well, we'd gone through that with CTE,
16:08
chronic traumatic encephalopathy. And that's these athletes who get
16:14
can cost even minor concussion. If they get it 50, 100 times, the brain starts to generate. Well, they saw the same thing happening Microglia were activated, the cytotoxicity was destroying the
16:29
brain and it was occurring in these same areas. And a number of these athletes were developing Parkinson's disease and dementia. And they looked at the brain. Well, we're seeing much of the same
16:44
pathology. We saw in a disease, we thought it was idiopathic. But now we know this idiopathic was caused by the trauma. And we know that trauma activates microglia and it produces exciting
16:57
toxicity.
16:59
So then the question was, well, what are these chemicals actually doing? And that's when I came up with the idea of immunorexcited toxicity, and when you look at this. Let me stop you there
17:11
because what you've, and we can bring everybody summarize everything up to date, what we saw was an image of the president who has a degenerative disease And then what you've talked about is that
17:25
you can see this disease in many other different forms, and
17:30
it's caused by, it
17:34
can be caused by trauma, you just talked about that many times. The boxers, football players get this, and they talk about Parkinson's disease and dementia. Everybody knows about that from
17:45
reading a sports page,
17:48
and the farmers who are using the toxins to the weed killers and so forth to take care of the crops and so forth. They wind up getting the same picture.
17:59
and then if you get toxins like heavy metals and aluminum, you get the same picture. So it looks like what you said back a little few minutes ago is there's got to be some basic common pathway here
18:14
that's activated, and then you get to the fact that these protective cells are microglia, which normally are like you get an infection in your arm, you got protective cells everywhere. But what
18:26
happens in the brain, these cells can become hypersensitized with repeated injuries, like if I keep getting the pesticide, or if I keep getting hit in the head, they're keeping being activated to
18:40
keep being in a repair state. And so now we've got these activated microglia, which are normally protective, and what you're saying is these are now have been accelerated to the point of almost,
18:54
it's almost like an audible immune response, where your cells are hyperactive and attacking you.
19:00
And so now here we've assembled a huge amount of evidence that suggests that there is this final common pathway and that's what you were just, I stopped here. And it gets to immunoexcitotoxicity,
19:13
which is a term that you put together a number of years ago. So why don't you go from there? 'Cause I think that one other thing is that you find how does it get in the body? Well, the toxins we
19:27
can figure you can get them by either you get on your hands, you ingest it, you get it in your nasal passages. Your nasal passages are connected right to your brain in the front here, and that's
19:39
where the olfactory, the smell senses go right there. And actually right next to it is the memory centers which is in the temporal lobes. So in the front part is a processing part of the brain. So
19:53
if all that gets affected, you can't remember, you can't learn, you don't know where you are, You can't speak, that's what we just saw.
20:01
And and with repeated repeated repeated injuries, it only gets worse So I think that's a brings us right up to here and you want to go further go ahead. Well, what you're seeing a Whole array of
20:17
chemicals and metals other things trauma What was the common denominator? Right. How could they all be connected? They're all activating microglia. They're all inflammatory And so when you look at
20:34
misfolded proteins like you see an alpha-synuclein Well, what causes the misfolded protein and inflammation?
20:43
Excited toxicity
20:47
And so these other so-called experts of the drug to flow shoulders and said we don't know it's just a mystery Well, it's not a mystery. You didn't look at all the factors. You'd like a detecting
20:59
you have to you have to examine, you have to be inquisitive. And you start looking and say, what puts this together? For instance, for years, I've worked with autism. And they thought it was a
21:13
mystery. Why are males more often affected than female? Well, look at the development of the brain. Males develop activated microglia in their brain very early. Females, it's much, much later
21:29
and they're not activated.
21:32
And that goes to priming. And priming says, well, if you take a microglia and you stimulate it, just a minor stimulant, nothing happens, except inside the microglia, it's producing a lot of
21:49
enzyme changes to make more inflammatory cytokine and exhale toxic. But it doesn't release them Then you stimulate it again.
21:60
And then it becomes an active microglia. Then it's really gearing up making these things. It's about three, four higher than a normal microglia. And then you stimulate it again, it releases them.
22:13
And so that part of the brain becomes highly inflammatory and it's secreting a lot of excitotoxic, which is destroying the parts of the brain that has to do with thinking, cognition, and memory and
22:29
learning.
22:30
It all makes sense, it all fits together. So then you begin to say, well, it's not that big of a mystery. And so you ask, well, how's that aluminum entering through your nose? Well,
22:43
geoengineering, they are spraying the skies with nano-sized aluminum and people breathe it. And it goes straight into the olfactory nerves into the brain and it's been traced It did radio tracing.
22:60
to see where it's going. They can see it go through the olfactory nerve and bulb into the brain. Why are they spraying the atmosphere with aluminum? What's the
23:11
reason? It's this crazy idea of global warming. They think it's gonna reflect the sunlight back out into the space with no consideration of what does now aluminum breathing and in the water and in
23:26
the food supply actually doing. Even though we've known since 1911 that aluminum was neurotoxic.
23:37
We've known alone that. Now why is nano aluminum more toxic than aluminum you find in nature? Well, it's due to surface area. Nano aluminum has far greater surface area and it penetrates deeper.
23:52
It goes inside the cell, inside the DNA. It goes everywhere Where no more. Does that interrupt the metabolic processes? They alone in them itself. How does it act or do we know? Well, it's an
24:08
inflammatory nitis. It's constantly producing inflammation which keeps their microglia activated. For instance, with a head injury, they found a man had a head injury. His microglia were
24:21
activated for 17 years. And they did the study on a group of autistic patients who were 40 years old, and they all talked to their brain, their microglia was still activated. So what you're
24:36
telling us is that there are a whole bunch of toxins in the environment, the pesticides, and that can set them off. But that doesn't mean you have to be, have the pesticides continuously
24:47
throughout your life. You could have aluminum, somebody would go be a fighter. You get all of those different things add up and do that. And you said the aluminum's in the air, but it's in the
24:57
water too And you write that, it's in the. It's in the water we drink, is that correct? Yeah, they add aluminum to the water as a clarifying agent, supposed to remove the particulate matter.
25:09
And so drinking water has added aluminum. Now, in England, as a researcher, who did research on this and found out you could produce dementia by drinking water aluminum
25:25
And there's a famous case by Cruz, actually, who I know,
25:32
and which there was a spill of aluminum in the drinking water, it was higher than normal. And people died and people had instant Alzheimer's disease. And when they looked at their brains, it
25:44
looked just like Alzheimer's disease.
25:47
Well, you mentioned autism disease. So it's obvious that disease that comes over a period of time in your life, Why do the kids. who are young kids who get this autistic disease, which is a male
26:00
development of the nervous system, probably with nervous system damage and so forth. And you know about this, you've written a book on it. Why do they get it early? Where do they get a dose of
26:11
some kind of toxin early, which activates all this? They're vaccinated. Oh, okay. And the number one vaccine adjuvant is aluminum, aluminum on drugs And it's different from ingested aluminum.
26:27
If you ingest aluminum, only a small amount is absorbed.
26:33
If you inject aluminum, 100 is absorbed. And the amount that these children are getting in the vaccine schedule exceeds the toxic level that should be in an adult. Oh my gosh. And how many
26:47
injections do they get through school? Well, it's up to 65 injection.
26:56
So. And then they continue it all the way through college. I mean, you go to college where you got a good new infection, new injection. You go to some high schools, you got a good new injection.
27:07
And so they're constantly being injected with aluminum that is 100 absorbed, producing these changes in the brain. And what one of the researchers, Dr. Bilbo, found is that these changes in the
27:23
brain will affect you as an adult. And she quoted four of my papers that I wrote written about what happens with this aluminum and what happens with mercury.
27:38
So you slated to study in Guam, or was there a study about
27:44
this? What did that study show?
27:48
Well, what they found is they were eating this sea
27:54
And they thought scikade was causing this, they call it ALS Parkinsonism Dementia. It's all in one person. And so Dr.
28:08
Chris Shaw, he studied intentionally and found out it was aluminum and the soil. The soil was real high. The aluminum in their motor cells was extremely high and it activated the microglia I was a
28:26
moderator at a meeting in Jamaica and Dr. Shaw was there and I asked him, I said, Duck, show, how many times have you seenmicroglial activation in
28:37
your studies with aluminum? So every time,
28:43
it always produces microglial activation in the spinal cord. Well, it does the same thing in the brain.
28:51
So we have a common path, we have a common agent, now we've got a pathologic study among others which shows that it accumulates, we've got all kinds of different ways it can enter the neuromus
29:01
system in addition to other forms that can activate the microglia and activate this common pathway through your life. So there must be a critical level you reach where it just begins to, the nerve
29:17
cells begin to degenerate Yes, that's what excited toxicity does is they were always, most of the articles in autism are written by immunologists. They don't know anything about excited toxicity.
29:32
Well, I had a friend of mine was an immunologist and he wrote a book about autism. And he studied all the papers that had ever been written. I mean, it was an unbelievable collection. He wrote
29:44
this book about what he found. In that, he said, Dr. Blalock has explained They explained all of it.
29:53
And so he looked at all the data. And of course, I looked at a good bit of the data myself. And what you're seeing is a whole array of things and the common denominator information. They all
30:07
produce information. They all activate the microglial.
30:12
And this priming is very, very important. Is if you look at the vaccine schedule, they'll give six to nine doses of the adjuvant in a single office visit. No pediatrician would do that to himself.
30:29
And this is a little child. Then a month later, they do it again. And then a month later, they do it again. And then a month later, they do it again. That's priming. What's happened is a
30:38
microglial keeps gearing up, gearing up, gearing up. And so as the studies of pathological studies showed, Well, the microglial were activated for 40 years.
30:51
And I had literally begged many of these researchers at meetings
30:58
to do microglial activation scanning. And they wouldn't do it. It was amazing. And finally, someone who I didn't know did microglial scanning of the autistic brain. It looks like the microglial
31:14
activation you see in Alzheimer's disease. All
31:18
the microglial in the brain are activated
31:21
So we see just using the aluminum then as an example of something that's really triggering the system. And obviously, if it comes in a large amount, that becomes a major trigger. If you're a
31:32
fighter, it's because you're getting these constant blows to the head, which is activating the same system, right? And what happens is if it's the aluminum, it goes into the astrocytes and which
31:45
are the structural cells in the nervous system, it goes to the neurons, take it up to.
31:51
Well, the neurons do, the number one accumulation of aluminum is in the astrocyte. Well, the astroite is the number one story site for glutamate. And if the aluminum gets high enough, the
32:05
astrocyte will die and release all of this glutamate. So now I think we're getting the time to explain immuno-excitotoxicity to the audience. So we've got the microglia primed. We're continually
32:19
stimulating it It's releasing a whole bunch of cytokines and all kinds of other things. Plus I would assume some glutamate and now glutamate, which could be a
32:29
transmitter, now a transmitting agent in the brain. Now it's just the cells are dying. It's being released extensively. What is it doing to the surrounding nervous system?
32:41
Well, it's extremely destructive. Glutomate has a neurotransmitter, so no one transmitter in the brain but almost all of it's inside themselves.
32:53
the neuron. Outside the neuron, it's extremely toxic. That's the purpose of the astrocyte and the microglia. They have transport protein. Their job is to remove it from the extracellular space
33:06
and put it inside the microglia and inside the astrocyte. We're safe. And what happens with all these conditions, whether it's trauma, boxing, inflammation, heavy metals, they release it. They
33:24
cause these microglia not to go to sleep, to stay active, and to release high levels of the excitotoxin, and pro-inflammatory cytokine. And what they found is if you combine a concentration of
33:41
inflammatory cytokine, there's too low to cause any destruction. And a concentration of glutamate it's too low to cause instruction if you need. mix them together, they're fully destructive. Okay,
33:56
so now we have a mechanism that can cause Parkinson's ALS. Now, I'm gonna just go off to a little side path here 'cause I talked about the present and the Lewy body dimension, which is really an
34:10
accumulation of misfolded proteins in the cell. And that protein actually, alpha-synuclein is responsible for the release normally of transmitters and so forth at the end of the nerve cell, correct?
34:25
Yes, and
34:28
the problem you have is that you take normal alpha-synuclein and you expose it to inflammation, it misfolds, then instead of being helpful, now it's harmful. This is just what happens to beta
34:44
amyloid. We all have beta amyloid, it takes a brain. under inflammatory conditions, the beta amyloid changes its chemistry and becomes highly destructive. Excellent. As an inflammatory.
34:58
Excellent. So now we talked about things that people, you read a little more thoroughly, you talk about beta amyloid, they want to see how much beta amyloids in the nervous system. Now we got
35:08
alpha-synuclein and in low bodies and they're misfolded proteins, but they're stimulated by what you're saying, this inflammatory overreaction, immuno-excitote toxicity, and then the glutamate
35:23
gets in there and it does depolarize the cell membrane and the cell keeps discharging. There are many ways the cell can die as a result of that. Correct? Yeah, what Dr. Olmey who discovered the
35:37
process of excitote toxicity, I was a guest at his house in Business Laboratory And he said, what happens if you expose the cell to. too high a dose of glutamate, not extremely high, but higher
35:53
than normal. Within an hour that cell would die, all the neurons will die. And so that you self this cascade of reaction, which involves a number of inflammatory concepts, like
36:11
PGE2, which is prostate gland in E2, and
36:16
it activates COX2, it activates a number of inflammatory processes to cause this change. And so people are blaming beta amyloid and alpha-synuclein, well, they just worsened the process. They
36:33
were caused by what caused the destruction. They're just part of the destruction. Now, let me ask you a question you alluded to it earlier. I just showed you, I went through and read a lot of the
36:46
things that the average person would pick up the internet and say Parkinson's disease from the Parkinson's Disease Association or Alzheimer's Association. Nobody talks about this. In fact, as I
36:58
quoted earlier, nobody knows the cause of it. Well, the answer is what you're saying, there is a cause. We do know what it is. What's the reason for this? I mean, why is there a big gap? Why
37:10
do these people know this? Well, what you're starting to see is, since I wrote this article for your journal, is there's a number of major researchers in the area of parks and isn't there or
37:23
quoting these sort of? And they even have paragraphs which discuss immunoexcited toxicity and have stated that the process seems to be immunoexcited toxicity. So it is changing. And these experts,
37:37
these researchers in the area of parks and are coming about. Like I said, Chris Shaw, and Vancouver, the neuroscientist, he said I'm exactly on target. He likes it. He thinks it's right. Goes
37:53
along with all of his research. And what he did and one of his elaborate experience, he took some rats and he injected aluminum subcutaneously. And then he examined their spinal cord. And he found
38:09
the anterior horn cells, particularly in lumbar and cervical area, had all the changes of ALS. And it was excited to talk to
38:21
you. So the nerve cells in the spinal cord you're talking about are the nerves that make the arms move and that legs move and so forth. Those are activated by neurons. Same is in the brain where
38:32
it's, you're getting all kinds of different connections that are doing things. And what he found is the same thing in the spinal cord is in the brain Why did you go to this final court?
38:44
Well, it comes from the GI tract. There's two ways that it gets in the spinal cord. One is through the nerves that feed the GI system. They go through ganglion and go into the spinal cord. The
38:56
other's circulating in the blood. It'll enter the spinal cord and brain through that way. So there's two routes of inflammation. There were some researchers, which I quote in my paper, who looked
39:10
at the progress of these diseases, particularly Alzheimer's and Parkinson's disease. And
39:20
what he showed is that they're going through steps. And he said, if you look at the brain, it's a gradual movement from this inter-renal area, hippocampus, frontal area, predoload, it's on a
39:35
march. And by the time you're totally demanded or dying, it's effective to hold brain same thing we see with ALS. it's just marching up to spinal cord, then you start seeing it in the brainstem,
39:49
then you start seeing it in other, and then some of these patients will come to domain II. And we used to think these were separate diseases. We said, well, there's Parkinson's disease, there's
39:57
Alzheimer's, and ALS. Well, in a Guam examination, you saw all three diseases in
40:05
the same person. And so they started looking at Parkinson's and they said, well, half of Parkinson's patients become demented And then they realized there's a cross-flow from all these diseases.
40:20
And we look at the number of people with Parkinson's who develop ALS, a fair number.
40:26
So they're not pure diseases. They're overlapping. And the only thing that wouldn't match this was immunogenetic, 'cause it's no respecter of organs.
40:40
Well, it's this terrific, terrific explanation for a very complicated dissettive diseases and problems that have similar manifestations. Now, I've, I'm in the audience, I'm saying, what do I do
40:53
to, what do I do to prevent this? Well, one thing that's obvious is, I shouldn't be getting, my kids shouldn't be getting these vaccinations, but you can talk about that in a minute. What else,
41:03
what else can I do to stop this from happening? I mean, it's, I almost can't control these things. They're spraying the air, it's everywhere, and I get the end of my nervous system. Is there
41:14
anything you can use to treat it? What do you, so how do you answer that? Well, avoidance is a big part of it. So, pesticides, herbicides, if you use them in your yard or whatever, you've got
41:25
to be extremely careful with them. I used to see people, I had their yard spray and little kids were out planning it. They're rowing it, their toys were in it, and they're putting it in their
41:36
mouth. It's on their shoes bringing in the house.
41:40
People don't think of these things, but they have found pesticides and herbicides in the stratosphere in the Antarctic.
41:49
There's no plants up there. Nobody's sprayed pesticides up there. So it's going all over the world. And we're using
41:58
tons of the thousands of tons of this every day all over the world. So that avoidance is one thing, when you can, as much as you can One thing is there are certain natural compounds that remove
42:12
aluminum from the brain and the body. For instance, curcumin is known to bind aluminum, and either it will remove it or it makes it non-toxic if it can't remove it. Querces didn't do that somewhat,
42:27
and trifla will remove some aluminum and it will remove flora
42:35
We have a number of compounds. Now are being discovered that are very powerful at removing these compounds. There's also anti-inflammatory compounds in these plant extracts. So when you start
42:51
looking at
42:54
bacopa, balkylene, when you look at apoganin, asperidin, they're powerful, powerful, and inflammatory. And so if you combine the two,
43:06
you're drastically reducing inflammation, you're lowering the level of aluminum.
43:12
And for instance, like nano curcumin, it will spread through the brain very easily, it either neutralizes the aluminum or removes it, and it removes it from the rest of your body. Well, I see
43:28
people all the time, they'll get a glass of water and they squeeze lemon in it or they get T and they squeeze limited. don't do that. Citric acid will make the aluminum increase its absorption
43:45
through the GI tract tremendously. Citric acid, malic acid, and glutamate. And what they found, and I talked to some of the researchers about this, is that glutamate will not only increase the
44:01
amount of aluminum in the brain, it increases the glutamate in the brain. So aluminum might not be absorbed very well, unless you're eating a meal that's high in glutamate. Then the brain level
44:14
will go up, and it goes through several areas of the brain, the glutamate will increase because of that combination of aluminum and glutamate. We also know that fluoride is highly reactive,
44:29
aluminum is highly reactive, and I have a research in the Czech Republic I wrote the book with and several articles. And she said, you never see fluoride that is not combined to aluminum. That it
44:43
always, the two of them will combine either in the outside atmosphere or in the water or whatever, but it'll also combine in the body. So we see in all these diseases, high level of aluminum
44:59
fluoride, the compound. And I wrote an article in your journal about it, the effect on the developing brain as well as the degeneration to show the mechanism of it. And she explained the effects
45:14
on the enzyme systems.
45:16
So really, I just have been a terrific review and revelation of what is the cause of a lot of these degenerative diseases. It's a lot of environmental toxins, as you started out saying, and it all
45:32
goes to some very basic common inflammatory mechanism then becomes an inflammatory mechanism that can be destructive and destroys the neurons and the cells and it gives you all these kinds of
45:45
different diseases, travels through the nervous system and it manifests itself in different ways.
45:53
Any other thing you can think of before we close because I think you left the audience with a greater understanding of this and I'm sure they're worried about, well we'll give them a list of the
46:07
compounds you mentioned so they know and obviously they can get it from your newsletter and this isn't a promotion of that but it's just an analysis of what the science is so that we can alter our
46:21
behavior to avoid these problems. Anything else you wanna add that we didn't cover? Well, other than removing the aluminum, there's also other compounds that are very powerful inhibiting
46:33
microglial activation.
46:36
Again, common vitamins, riboflavin,
46:40
fibosphate, fibodoxal fibosphate, thiamine. And recently they found that people even, who've had Parkinson's disease for a decade, if they take high dose thiamine,
46:51
they can completely reverse. And thiamine, deficiency, and what I found years ago was that everybody thought this destruction at the
47:02
base of the brain was due to thiamine deficiency directly in what it showed was actually the damage is excitotoxic
47:13
by amine inhibits excitotoxicity. And so there's a double thing. And the other thing is don't have your food in aluminum. Don't put aluminum foil on it, don't cook on aluminum, don't have
47:29
aluminum pan with aluminum exposed, And don't squeeze a lemon in your. your drinks. And these things will lower your loom 'cause what happens with food, it'll extract the aluminum, particularly
47:43
if it's acidic. So if you cook an aluminum pan or you wrap your leftovers or something and aluminum, it will draw the aluminum out and put it in the food and then you're consuming. And what
47:59
happened is over a lifetime, you're consuming that, that's what I put in the article in your journal, is that you've got a lifetime of a oral absorption and a vaccination, things like a flu
48:12
vaccine every year.
48:16
And just a terrific summary and analysis of this problem, Russell, way ahead of your time as usual. I think the audience is gonna get a lot out of this and
48:30
I can't thank you enough for taking the time to inform us about this, tell us about it in a way that's understandable.
48:36
So that's all I will be back with another installment of the the
48:43
nutritional advice and thank you very much. Okay. Thank you, Jim. I appreciate the opportunity Okay, well.
48:58
This talk that you've heard and discussion
49:05
is focuses on the concept of immuno-excitotoxicity
49:11
with microglia, which are the
49:15
immune protectors of the nervous system, the white cells of the nervous system that are activated by multiple, multiple toxins, stimuli, and threats during a patient's life. And
49:31
these activated microglia then after a period of exhaustive stimulation release glutamate
49:40
and cytokines, many of them, you can see listed there, they produce free radicals, reactive nitrogen species and reactive nitrogen species.
49:56
which then can enter the mitochondria and cause dysfunction in the metabolic pathways there, leading to the cell death in a concept he entitles immuno-excitotoxicity.
50:15
In future talks, Dr. Blalock will go into more detail about the biochemistry of these pathways, which are essential to the understanding of this new concept of the cause of neurodegenerative
50:32
diseases.
50:36
These are the key references I've mentioned. You should take a screenshot up because in these papers, you will find all the references you need to further pursue this subject. The
50:50
biochemical basis of neurodegenerative disease, The reference is given at the bottom.
50:58
Parkinson's disease,
51:01
again with the references at the bottom,
51:04
additive aluminum as a cause of induced immuno-exciter toxicity, which becomes a hallmark of how this disease can occur with this and other stimuli which are become toxic
51:21
and the treatment of autism spectrum disorders
51:27
So take a screenshot of this information so you can have it for your reference.
51:33
We hope you've enjoyed this presentation.
51:38
The material provided in this program is for informational purposes and is not intended for use as a diagnosis or treatment of a health-related problem or as a substitute for consulting a licensed
51:54
medical professional.
51:58
Please fill out your evaluation of this video at the end of the program to help us improve the information we're bringing to you in SI Digital.
52:07
This recorded session is available free on SI Digitalorg.
52:13
SI Digitalorg, it's oars
52:16
in your comments and questions, or requests for CME credit to ossmansidigitalorg There are many ways to learn. Surgical Neurology International is a 2D internet journal,
52:26
like most others. It's editor-in-chief is Nancy Epstein. The web address is SIglobal. SI Digital is a 3D video journal. Interactive, with discussion of the subject presented, so that you
52:53
have more ability to learn the most you can about it. The web address is snidigitalorg.
53:03
Both of these publications are free on the internet 247 365.
53:15
Surgical Neurology International is read in 239 countries and territories and is the third largest neurosurgery journal in readership.
53:27
SI digital innovations in learning is now read in 100 countries in four months after the publication was initiated, it's the first video journal of neurosurgery
53:41
Our goal is to help people throughout the world.
53:46
The Medical
53:48
News Network was designed to bring truthful medical and science news to the world
53:56
The program is supported by the James I. and Carolyn R. Osmond Educational Foundation. It was copyrighted in 2024. All rights are reserved.
54:13
Thank you very much for watching