0:02
SNI, Surgical Neurology International, an Internet Journal with Nancy Epstein,
0:11
etc, and SNI
0:15
Digital, a new, editorially curated
0:21
neurosurgery and medical information multimedia platform with operative videos, expert interviews, podcasts, and global active, interactive discussion of information
0:38
for the next generation of clinicians in 13 languages. With James Osmond as the editor-in-chief are pleased to present another in the SNI, digital investigative series
0:56
on Dr. Blalock reports the metabolic basis of cancer. A summary.
1:07
Dr. Blayox, the CEO of Theoretical Neuroscience Research, an LLC and Associate Editor-in-Chief of the Neuroinflammation Section of Surgical Inter-Net, National Neurology, International and SI
1:20
Digital, he is a board-certified clinical nutritionist, and the creator and editor of the Blaylock Wellness Report, author of multiple books, scientific papers, he's a health commentator on radio
1:35
TV in the epic times. He's written books on natural solutions for liver disease, cancer patients, solutions for natural health, excitotoxins, which is part of the talk he's giving today. And
1:53
with Dr. Osmond as the associate editor of the China virus, what He publishes the Blalock Wellness Report, which is a monthly nutritional newsletter published for over 20 years,
2:09
and these are examples. One is cancer is actually a metabolic disease. The subject of today's talk and natural compounds that offers more benefits for cancer treatment and a book on natural
2:22
strategies for cancer patients. You can subscribe to the Wellness Report at newsmaxcom
2:30
Hello again, I'm Jim Ousman. And I'm here with Roso Blaylock. Everybody knows Roso Blaylock. Well, he's made major contributions to SI digital.
2:42
Many of them focused on the metabolic basis of diseases. And he has almost 20 videos on SI digital, covering various aspects of this, including what happens with microwave radiation And if we have
2:52
time, we'll get into that.
3:01
but today he's going to talk about something that he's had three previous videos on in the last a month. And it's on a subject that's very controversial. And the subject is, what is the cause of
3:15
cancer? Is it all these genetic changes that everybody's talked about that everybody read all these papers in the literature, all these summaries, all these reviews, that's what it is, are all
3:28
genetic changes. And he's about to tell us, and he told us on the three previous videos. The first one said, Cancer is a metabolic disease. And then the second one had to do with what are the
3:42
various agents, you know, smoking and lung cancer, you know, a variety of other things, toxic chemicals with the
3:56
insecticide agents can do that. so far. So we know that there's a relationship. And then lastly, the final video that he had was that basically, in most of the cases, cancer is related to
4:14
viruses that are latent in the cells activated and produced a programmed development of a cancer cell, particularly targeting it on stem cells. And these are all thoughts
4:34
that are not in the common textbooks. They're not, they're not in the common, common knowledge. Many doctors don't know this. So again, what we're Dresses is doing is interviewing, is
4:43
introducing us to controversies in medicine, which we want to present on SNI digital, things that you didn't learn, or you didn't learn completely, or we're not correct, and why they are not
4:56
correct, and what is the correct answer. And so today, I got a whole bunch of questions about the three videos on cancer. And I'm gonna ask the questions that came up to Russell so we can explain
5:12
all this so that you can take away from hopefully this 45 minute video, a concise summary of what is the cause of cancer and its metabolic and its likely viral And those are shocking, shocking ideas
5:31
to most people. But our goal is to take on controversial issues and tell the truth. So Russell, let me ask you the first thing we did in the first video. As you talked about a doctor who was
5:45
writing papers back in the early 2000s, 2006, 2014 on Thomas Seifried and he did a series of seminal experiments. that are fundamental to understanding why cancer is a metabolic disease. Can you
6:03
briefly describe those for our audience 'cause they are absolutely significant in understanding this?
6:11
Yeah, thank you, Jim. Basically, Safer looked at the history of pathology of tumors and he said, What's strange is that you see tumors in which there's no genetic defect You see tumors have
6:26
completely different genetic defects, yet they're all producing the same cancer. And so - As you said, the major question was, what's driving
6:41
this disease? Is it metabolic or is it, as we've been told, a genetic disease? So opportunity appeared some time ago in which they did nuclear DNA
6:52
transfers nuclear your take
6:54
out of the cell and normal cell. and take a cancer cell nucleus and insert it in the cell. Well, if it was driven by the mutated DNA, it should convert that cell, daughter cells into cancer cells.
7:15
So they inserted the nucleus of the cancer cell into the normal cell after removing its nucleus And lo and behold, the
7:29
daughter cells that originated from this all seem to be normal. They're not driven by the mutated DNA to produce cancer cell. Well, to further emphasize that, what they did is sort of a reverse
7:47
system in which they took the cells and they took a cancer cell, and they put the nucleus of a normal cell in
7:60
it and then they went even further, they said, well, if it's being driven by something in the cytoplasm, most likely it might have contrem, let's put
8:13
a cancer cytoplasm, remove the nucleus that is highly mutated, put in a
8:23
look at the daughter cells and see if they're cancerous. And so what they found was all the daughter cells were cancerous, even though the nuclear DNA was now normal. And so they concluded from
8:38
that, well, it must be driven by the mitochondrion, the mitochondrion is defective, and that the cell is no longer producing normal cells, it's producing cancerous cells.
8:54
transfer experiment, they repeated it throughout the time period until present, and each time they found the same thing. If you transfer a cancer nucleus with all these mutations in it, and the
9:10
cytoplasm that was normal, and the mitochondrion that was normal, it produced normal cells. But if you did the situation where you had
9:23
a cancer cytoplasm, in other words, the defectives mitochondrion, and you had a normal DNA, normal nucleus, it produced cancer cells. And so it demonstrated that it's not being driven by the
9:41
nuclear DNA, it's being driven by the mitochondrion being defective And, of course, this goes back to Warburgs.
9:53
demonstration. In Germany, I think it's four back to
10:01
2008 or something like that, and what he demonstrated, all cancer cells are not metabolizing normally. They're metabolizing
10:10
without the use of the mitochondria and only partially with Krebs cycle, which is part of the mitochondria in the outer memory, and that most of the energy and biomass is being produced by
10:26
glycolysis, and that the entry of glucose into the cell is dramatically increased in this case, and even in the presence of oxygen, which normally, in a normal cell, would take the glycolysis as
10:45
it reaches peruvate, and have it enter a Krebs cycle, go through the cycle, and produce
10:53
the
10:56
substrate or the electron transport system. In other words, you've got a three metabolic systems. And the electron transport system would produce most of the energy. But the cancer cell stopped at
11:09
glycolysis and only partially utilize Krebs cycle and that the number one substrate was glucose And then they found, well, it's actually also glutamine.
11:30
The amino acid, ubiquitous amino acid. And the glutamine enters the Krebs cycle and is converted by glutaminease and enzyme into glutamate. And then the glutamate enters the Krebs cycle at
11:47
alpha-ketoglutamine.
11:49
And so it's producing some of the energy and some of the biomass for production of cancer cells.
11:58
It's recently been found that the glutamate also is released by the tumor. It's in the tumor microenvironment. It's acting through the receptors, just like normal glutamate does, and that tumors
12:13
that have higher glutamate are more metastatic, more invasive.
12:20
And so if you look at melanones, you look at glioblastones, you look at megaloblastomas,
12:26
the higher the glutamate release, the higher the content of the
12:32
glutamate, and therefore the higher the glutamine, which is converted to glutamate,
12:37
the more aggressive it is, the more deadly it is. This is true in breast cancers, colon cancers, all the cancers they've examined, is that the higher the glutamate, through these two mechanisms,
12:50
the more aggressive the cancer.
12:54
Let's take a minute to look at
12:58
some of these diagrams that you made. We'll go to this one first here. So this is what you're saying is happening. The glucose comes in the cell under Warburg's hypothesis, right?
13:13
And glucose goes to pyruvate
13:17
Normally it would go down to lactate, but it goes to pyruvate. And so it's bypassing the pathway where it goes into the cell and it's metabolized and informs energy. Is that, did I say that
13:33
correctly? Well, normally the glucose enters is converted through a series of enzymatic changes to pyruvate. And the pyruvate is converted to acetyl-CoA and energy. the mitochondria. So here's,
13:50
here's this to help explain it better. Here's our glucose, glucose, phosphate, fructose, and
13:59
pyruvate. And normally, it would go into the, normally it would go into the cell and be appropriately metabolized, right? Right. You see the, the probate inside of the mitochondria is converted
14:13
to single CoA, and that has to do with lipid metabolism and operation of the tricyclic cycle, the
14:26
Krebs cycle. And so that, that's a normal position. Now, what happens is this is so
14:33
unusual in that, in the presence of oxygen, that's the path it takes. When oxygen is not present, the peruvate is converted to lactate, which
14:45
is the nacic. And so, the microenvironment becomes acidic. And that's what we're seeing is that most of the probate is converted to lactate, even in the presence of oxygen. And it's only a small
14:60
part is entering the Krebs cycle inside of the mitochondria, the membrane, outer membrane of the mitochondria.
15:10
But the tricyclic or the
15:13
Krebs cycle is not working properly. So what happens is this all gets its energy by taking glutamine, converting it to glutamine, it gets back into the cycle and then it produces energy in the
15:28
different components. Is that right? That's right. It becomes dependent on the glutamine. And so glutamine becomes a major source for energy to drive the growth of the cancer rather than just
15:44
glucose. So together, the two glucose and glutamine is driving the cancer to produce energy and its biomass to produce more cancer cells. And some cancer cells, as you see here in the MIC, which
16:01
M1C, which
16:04
is a gene, it will produce a dependence of the tricyclic cycle here, the Krebs cycle,
16:15
it makes it dependent on glutamine. And we call that
16:21
glutamine or lysis. And so glutamine becomes a major fuel for those kinds of cancer.
16:30
There are actually two fuels that are driving the cancer. Is this called the Warburg effect?
16:38
Well, this was the completion of the Warburg, the first part of the Warburg effect was the In fact, that most cancers were not utilizing the Krebs cycle completely and did not realize the electron
16:52
transport system. They were using fermentation of the glucose and it stops at lactate. And so lactate begins to build up in the microenvironment of the tumor and it becomes acidic. And that's why
17:09
all cancers are acidic
17:13
So is the defect here somewhere around this point here where we're converting perovate to a lactate instead of it having going into the, the crop cycle, is that correct? Yeah, normally when you
17:29
exercise and you're not getting enough oxygen to meet the requirements, lactate begins to accumulate and that's why your muscles get sore and you have cramps and these things but as oxygen increases.
17:42
The lactated were back to prove A and prove A to create cycle. And so the operation of the mitochondrion completes it all the way through electron transport system. It gives you all the energy you
17:55
need. It provides
17:60
38 ATP's, whereas just the glycolysis only supplies two.
18:07
So what the cancer does is it increases glucose entry into the cell in such high content that it's making up for the energy loss by utilizing glycolysis. And that's what the Warburg effect is.
18:25
OK, so
18:29
is it understood where there must be some biochemical change here that shifts it from going to make lactate, from making perovate to going to make lactate. Is that correct assumption? That's right.
18:44
Does anybody know what that is?
18:48
Well, if you read
18:51
cyphrid and you read the literature, you find out nobody really knew why this was occurring. What is altering this metabolism so it converts to glycolysis, even in the presence of adequate oxygen.
19:09
And that the mitochondria is not working properly An electron transport system's not working or at all. Nobody knew what's bringing about that transition, that
19:22
trigger. What has been discovered recently is that a virus, like the cytomegal virus, will produce the Warburg effect exactly. It seemed to be the trigger
19:37
So that's the link between this and the viral cause. of cancer in the latent virus in the cell, right? Right, the latent virus by itself can produce the warburg effect, warburg metabolism in the
19:53
cell, just like we see in cancer. And by the mitochondrion and it infects stem cells
20:04
and infects the immune cells. Big question that was always there is the unconscious said, well, we looked at a number of cancers, we either found no viruses or we saw only a few of the cells had
20:17
viruses in. And so that wasn't the cause of the cancer. Well, the modern research had looked at it again and said, well, those studies were severely flawed. The methods they were using to detect
20:32
the virus were inadequate. Even the PCR cancer does not show the oncogenic viruses The second thing was that. these late viruses, these anco-modular viruses, produce hundreds of proteins, and
20:51
those proteins, each one does a different thing to the cell, and what it does to the cell is everything we see in cancer, everything, and yet the mutations that we see that were always thought to
21:05
be the cause of cancer didn't produce a lot of these things, but the proteins produce every one of them. And so as we began to look deeper in that, and they repeated the experiment of modern times,
21:19
they found the same thing. Well, as we used more refined techniques to find these viruses, they found more, and that they were indeed in virtually all the tumor. And if you look at glioblastoma
21:31
multiforme, which is the
21:34
most malignant brain tumor, almost 100 of them have the virus, the late virus. If you look in the blood of the person who has a
21:45
glioblastoma, 80 has a viral. So as you begin to detect the virus, you're seeing almost virtually every glioblastoma has this oncogenic, oncomodulatory virus in some kind. Usually it's a
22:00
cytomegal virus. Then they started looking at prostate cancer. Virtually 100 of prostate cancer say it. And they looked at breast cancer The same number of breast cancers had it. They looked at
22:14
mucous salivatory type cancer. They had the same virus in
22:20
it. And so then you begin to look at this and say, well, in order for all these people in the world to have cancer, it has to be a very common late virus. Where you look in the United States,
22:33
over 60 of people have this late virus And they carry it for a lifetime.
22:41
certain things don't happen, like exposed to carcinogenic agents, it never wakes up and you don't know you even have it. And then they looked at other societies across the world and they found a
22:53
hundred percent of their society was infected. And then they found as you get older, the immune system begins to degenerate somewhat. So it's not as accurate. The latent virus begins to wake up It
23:08
tends to infect senescent cells, which are aged cell, and it turns other normal cells into senescent cell, which again wakes the virus up and produces the cancer, explaining why cancer is more
23:23
common as we get older.
23:26
Then we started looking at other things we know cause cancer, like radiation, UV light, carcinogenic chemicals. And we see all of them wake up these late varsity and can produce cancer. by waking
23:40
up to latent carcinogenic virus. So as you start to put these points together, it's all pointing to the fact people are carrying around these viruses, and they don't know it. It's not producing
23:53
any disease until they're exposed to these agents. Now we even have an exact answer. Why are these agents causing cancer? The causing cancer cause so many people or carry in the latent virus And it
24:08
infects the brain, the pancreas, the liver, all organs, kidney, bladder. And so we're getting a look at it and we look at breast cancers. Well, they found the cytomegal virus in the breast
24:21
cancer. And when they looked at the particular type of cancer in the breast, they were in different locations inside of the breath. And they produced many of the same changes we see of the cancer
24:36
of the breast.
24:38
When you start looking at turbo cancers, which we're experiencing today with the COVID-19, we're seeing that it's not only affecting all of these things we're talking about, it's also affecting the
24:50
immunity even worse than it is by the late virus. So the late virus can suppress immune system. When in 1920, Dr. Warburg, a German was looking at cancer, he studied it very carefully and he
25:03
said, the metabolism of a cancer cell is different than a normal cell. We know there's three layers of metabolism in the normal cell. You have glycolysis, which produces two ATP, and it takes
25:16
glucose and it passes through a short number of biochemical reactions and ends up with
25:24
probate. The probate then enters the mitochondria and continuously at reaction goes through the Krebs cycle, that produces electrons and then goes through the electron transport system You found
25:36
there was a defect. in the mitochondria, so that they prove it, now goes to lactic acid because it can no longer go through the other two processes. And the higher the lactic acid, the more
25:49
malignant the tumor is. And it found the higher number of cytomegalovirus in the tumor, the more malignant it is. And more aggressive. And the more likely you're not gonna be cured of it. And
26:03
this was true for medulla blastones, it was true for glioblastones, it was true for colon cancer, it was true for breast cancer. So we're seeing all the things. The more of the latent viruses in
26:16
it, the more of the proteins produced by the latent virus that are in the cancer, the more aggressive it is, the difficulty of trying to get it to respond to any treatment goes along with this.
26:31
And so it's answering virtually all the major questions we have. Well, then I looked at P53 gene, which is a suicide gene. Its purpose is that when a cell gets damaged, it's a nuclear DNA
26:47
gets damaged so much, the P53 kills the cell, so it won't be converted to a cancer. Well, then they found P53 is deficient, the cell converts to war works metabolism.
27:03
So in all of these cases, we're seeing the mitochondria is abnormal in cancer cell. And if you look at the brain cancer, it has no crystalline inside of the brain cell, which are where the
27:16
metabolic electrons were changed doing its operations. And it's completely abnormal. They've never looked at the cancer, which had normal mitochondrial function.
27:31
And so if you take the mitochondria from a normal cell and put it in a cancer-saled torsion normal. So once again, now it can carry out its metabolism normally, and that prevents the cancer.
27:47
So all these questions are starting to be answered by looking at the metabolism of the cell and the fact that it's the carcinogenic virus, the latent virus, that is altering the metabolism of the
27:60
cell. And if you look at some of the proteins it produces, it produces a warborne effect If you take a normal cell and infect it with the cytomegoid virus, it has the same metabolism as a warborne
28:13
cell. Let me ask you a couple of questions about what you just said. You mentioned that mitochondria in a cancer cell, mitochondria is abnormal. Is that just abnormal in structure, or is it has
28:30
mitochondrial DNA, or is the DNA is a DNA abnormal or what is abnormal in the mitochondria? We never talked about that before. What's the admin all about it? Yeah, if you look at mitochondria
28:42
inside of a cell that's carcinogenic, it looks structurally abnormal. It's a normal, I have a Christie inside of a, they're remembering of a mitochondria. That's where the electron transport
28:56
chains work. Well, it's absent and almost all cancer. Like mitochondria's deform You get bio-genesis of mitochondria in a normal cell. That is, it's reproducing itself. Or it doesn't happen
29:10
anymore. Some cancer cells don't even have mitochondria at all.
29:16
Some have a drastically reduced number of mitochondria. But in all of the mitochondria do not function normally and the structure is not normal
29:30
is I don't, maybe we don't know this answer. but if people looked at the mitochondria DNA to see other different cancer cells than they are among other cancer cells, which cancer cells and normal
29:43
cells, or has that not been studied yet? Well, if you look at all cancer cells, they're all abnormal. No kind of cancer. Brain cancer, pancreatic cancer, breast cancer, colon cancer. They're
29:56
all abnormal If you look at people that have mitochondrial defect, well, they can partially utilize the mitochondria. It's not like a warburg effect. It's not like a cancer, a mitochondria,
30:12
because the mitochondria is not working virtually at all in the cancer cell. And as the lactic acid begins to build up and the cell becomes more acid, and it's a dotic, it becomes more aggressive
30:27
If you reduce their lactic acid, it becomes less aggressive. Okay. Let me see what else. Now there's a couple of things that I remember, we talked about it before. When I was at the NIH, we
30:40
worked with SV40,
30:45
which a woman at the time, I can't remember her name was, had used to induce appendomoblastoma, it's in mice. And
30:54
here was at the time, the thing that was so striking about it, is here was a virus, you injected, and injected actually into the brain tissue And
31:08
the animal's forming, it's very high percentage. Okay, I don't remember if it's 100, but I think it was close. Get appendomoblastomas. Here's
31:13
a direct example of a virus. Injected, normal brain, animal comes up with a tumor. So there's something else to suggest there. There are other viruses, I would guess to do that. I'm not that
31:30
familiar with it, And, uh, And so that would be a direct example of what you're saying, correct? Right. And when they looked at the cytomegal virus in a newly born life, it increased the growth
31:46
of the external granular layer. Well, the external granular layer in the cerebellum is what formed the medioloblastoma. When they blocked the TNF alpha, it didn't happen
32:00
Well, if it was due to a genetic defect, it shouldn't have made any difference. And so all the medioloblastomas have started to make a virus in it.
32:13
So what we've done just discussed in these past few minutes is the fundamental principle, and that is cancer is a metabolic disease You've shown that by the fact that Dr. Seifrieder in his
32:29
experiments where you put his. a normal cell nucleus and a cancer cell
32:39
cytoplasm, and it'll become, it'll grow to be a cancer, but if you take a cancer cell nucleus, put it into a normal cell,
32:48
nothing will happen, but if you put it into a cytoplasm from a cancer cell with a normal nucleus, that grows to be a cancer cell So it's in the cytoplasm. And what you're saying is it's a lot of
33:05
proteins and factors are made. And we know that the mitochondria are abnormal and they have mitochondrial DNA.
33:16
We're not so sure what that is, but that's what we know. And we also know and you introduced us to the fact that there is, and it's found commonly, this was found, I think 1920 was Arta Warburg.
33:30
You've got to know Paul Price for this. that the metabolism of the cancer cell is changed. It's changed from diverting the normal glycolysis pathway right through the mitochondria, and it's
33:43
absolutely derailed, and it goes to produce lactic acid, perovate and lactic acid, which produces an acid environment, and then it can lead to other changes also.
33:57
Did I summarize that correctly? That's exactly right Okay, so we wanted to tell the audience, okay, this is the fundamental experiment that says that cancer is a metabolic disease. If you were to
34:13
assume that all of these cancers were related to some genetic change, you would have to assume for them to produce a cancer cell, which goes down a predicted pathway. It makes certain substances
34:28
that turn off the immune system certain growth factors for blood vessels and so forth. There's a predictable series of events that occur with a series of proteins that are made and they have to come
34:39
out in a certain sequence to do this. And if this were true, how do you get that from if it's a genetic cause and
34:51
randomly they're supposed to produce this end result? And it doesn't make sense. It doesn't make sense. There has to be another factor that begins to organize this so that you get the sequence of
35:05
events that occur. And what was known before is that you could take a normal cell, a completely normal nucleus, a completely normal cytoplasm, and you could change it into a cancer purely by
35:21
altering its metabolism, having no mutations whatsoever. All you do is alteration with capitalism. It becomes a cancer cell. Key piece of information, key. Okay, that's great. Now, the next
35:36
thing we did in video number two is that there are a number of things that are factors that
35:44
we know are associated with cancer. We haven't gotten to how the cancer makes this happen, which is what we're gonna talk about now. But for example, microwave radiation, if you take your cell
35:56
phone, that's microwave radiation in this cell phone. And we talked about the Havana syndrome before work. It was documented that the Russians had targeted our embassies with microwave radiation.
36:12
These people came back with neurologic damage. So the radiation produced damage. Damage is damage results in inflammation, okay? And we'll come to that Okay, we know that certain chemicals, some
36:28
toxins. do that. We know that mercury, we know that aluminum, we know that fluoride, right? These things have taken in excess, and you've made very good arguments that they are in excess in our
36:45
environment and in our body. The water is fluoridated, for example. These can lead to cancerous growth, okay? That's number, those are two. Radiation, radiation, and we see this, and we see
36:59
it in neurosurgery, radiate the brain, and what happens is they come back years later with a secondary cancer. It could be a meningioma, and everybody is wondering about it. They say, Well,
37:13
it's got to be related to the previous radiation, or they get skin breakdown, and they go to cancer there. So, radiation is another cause. What are some other causes? Well, like I said, or
37:25
besides,
37:27
they can cause the smoking. any kind of industrial chemical, many of them can cause it. Lead, mercury, they all result in it. But if you look at every one of these things that we know are
37:42
carcinogenic, they all activate these virus. Every one of them. They all activate the viruses. Every one of them. Okay, now do they also, so now we've covered that. Now do they also activate
37:59
the immunologic excitotoxic system? In other words, this inflammatory system
38:08
that produces an inflammatory cascade,
38:12
is that essential for this to happen? Well, it is because if you look at the end point of this, when there's a lot of study on this now, is that once you stimulate the immune system, that's why,
38:26
once it produces the inflammation. The information is directly connected to the glutamate system. And so all cancers have high glutamate in them. If you look at the emotional leg neck glial
38:40
blastomas, it's the ones that secrete the highest level of glutamate. Look at breast cancers, you look at coloring cancers, the ones that produce the most glutamate, emotional ligament. Well,
38:52
they first assumed, well, we know that there's two fuels for cancer One is glucose, now there's glutamine. And glutamine is converted into cancer cell into glutamate, by an
39:09
enzyme, glutaminease. And so there was a direct
39:13
connection in terms of its metabolism, because the glutamate, then, in
39:18
the cell, will enter the Krebs cycle and an
39:24
alpha-keton-glute rate. and produce the bowel mass per cancer cell. Even though it's defective, it still produce the bowel mass. Well, now we know if you take a triple negative breast cancer,
39:41
for instance, which is one of the worst of the breast cancer, and you just take a normal breast and you inject a cytomegal virus, it will become a triple negative breast cancer. You didn't do
39:54
anything else You didn't radiate it, you didn't expose it to chemicals, any of these carcinogenic ages, all you did is infected with cytomegal virus, and it will become a triple negative breast
40:05
cancer. Okay. And we know if you take all the other cancer breast cancers, ones that are sensitive to estrogen like that, they also have a cytomegal virus.
40:16
And we know that they connect directly to the estrogen of the prosthetic.
40:22
So the reason why we have to consider that the, what you've called, and you named us 10, 20 years ago, immuno-excitotoxicity. And somebody else had contributed to this idea. But
40:36
immuno-excitotoxicity is a fundamental response in the brain, but in the rest of the body in response to injury, toxin, or damage. In kind of information. We now have proven that We know that.
40:54
And you did some work earlier, two, 20 years ago, 25 years ago, you wrote a paper with Joe Morone, and it was everybody's wondering why the football players would come down later with Parkinson's
41:08
disease. And you wrote a paper together, which said that basically what was happening is that continued head trauma was producing continuous inflammation with a hypersensitized immune system in
41:25
microglia and the brain, which essentially would overreact every time there was more and then create a whole bunch of these transcription factors and
41:38
various signaling messages that eventually resulted in neurologic damage. Is that correct? That's right. It resulted in
41:48
exyloplaticisidin And when they looked at the brain, they all had all the attributes of exyloplaticisidin.
41:55
And what I was going to say about the glutamate in these tumors, because they all produced the glutamate, is the first we thought it was just a fuel. Then they found out if you take the glutamate
42:06
and stimulate the glutamate receptors, which are all kinds of cells, it drastically stimulates aggressive thyroid cancer. If you block the glutamate receptor, many cancers will become much more
42:21
safety, the chemotherapy and treatment.
42:25
Okay, that's important, another key item. Now, another point is you've talked about this before. I know you've talked to me about this. We have, there's a homeostasis in the body and
42:39
we have some excitatory effects in the body and this is basically because it's the most prolific or most abundant transmitter is glutamine, glutamine, right? Glutamine, glutamine. And what's the
42:52
inhibitory transmitter?
42:58
Redgamma. Gamma-meo,
43:02
reduric acid, yeah. Gamma, right, okay. Yeah, but it's not. And so, we wanna establish here to the audience, there is a balance constantly in everybody's body between these excitatory factors
43:15
and the inhibitory factors and when the inhibitory factors. when the excitatory factors, which here are largely transmitted by glutamate, and glutamate receptors, which are everywhere in the body,
43:28
everywhere. Then we have this imbalance, and we get the excitatory effects, right? And if we get too much GABA, we probably get the organism that's toned down, and so forth, and repressed to
43:45
some degree. And so now, another factor is the right time to do this We've changed our diets, and the diets now contain a tremendous amount of sugars, which lead to obesity, which is at least 50
44:01
of
44:05
the people in the world. This is, you're talking about an epidemic, that's what it is. And so, what happens is,
44:20
We take a lot of glucose in in order to make the food tasty. They add glutamate as a sweetening subject that makes it a substance that makes it, oh gee, that's really good. I want more of it,
44:37
right? It's a taste enhancer. Yeah, and
44:40
so what happens is now we're flooding our body. And we said one day there was 37 trillion cells in the body And you're ingesting that it's going everywhere in the body. And so there's an excess of
44:53
the glutamate now, which is abundant. And against this background of trauma or more stimulation and so forth, it just sets it up for these series of reactions to happen. Is that correct? Right,
45:09
and there's two basic things we're dealing. One is the immuno-exciter toxicity It was all through your album, Parkinson's, ALS. virtually every neurological disease, brain trauma, strokes.
45:23
Then we're saying cancer is also increased by dramatically. And if you look at melanomas, melanoma secrete a lot of glutamate. To hire the glutamate, the more aggressive is the cancer, the more
45:38
metastasis, the more lethal it is. Now we're finding all these things are based on the amount of glutamate
45:48
So now we're back into the metabolic basis of disease, which you spent your whole life on. You write a monthly newsletter for your 25 years on the subject and say, You got interested in this before
46:01
you went to medical school, and you've been studying this for years.
46:06
And so now we have some of their dietary change in the obesity which appeared in the world. Now we're flooding our bodies with glutamate. in addition to all these other kinds of things that happen.
46:18
And one thing that you didn't mention that you've written a lot about is autistic disorders, which is another nervous system disease, essentially which destroys the neurons. That's right, it
46:32
causes abnormal development. And this is one thing that connects the embryology, the development of a person to the cancer. And there was a theory at one time that all cancer are blastocystic,
46:44
that is they're a tropoblastic, they're
46:48
scattered primitive cell throughout the body that never mature and they become cancer. They become like an embryo. And so when you look at immunoexcitotoxicity in the case of autism, what's
47:03
happening is causing abnormal development of brain because glutamate guides the neuron you have philipolia on the neurons and it's guiding. Well, it's guiding the cancer cell just like that. And so
47:19
some people say, well, we look at cancer. A lot of the things that are occurring metabolically in the cancer cell also only occur in embryo.
47:29
And the other thing we haven't talked about is the calcium oscillation. And you look at all brain development is based on calcium oscillation. Well, what causes calcium oscillation? It's the
47:45
glutamate, there's a certain type of glutamate receptor called NMDA receptor that is connected to a calcium pore. It calls calcium to pore to the cell. Well, what controls the calcium amount in
47:58
the cell? The mitochondria. Well, if the mitochondria is defective, it's no longer controlling the calcium. The calcium is causing abnormal oscillations and therefore a lot of the factors we see
48:13
with cancer. particularly invasion and migration.
48:18
Excellent points, excellent points. So now we've talked about A, when number one, cancer is a metabolic disease. You can't have all these chromosome changes, all these genetic changes and
48:31
randomly occur to produce what is a very common pathway toward cancer in virtually all the cells that have it. Number one, and number two, we found that there are a variety of toxins in the
48:45
environment, all of which cause damage to a whole variety of cells. They all cause an inflammatory reaction, an inflammatory reaction with excitotoxicity because all kinds of transcriptional
49:00
factors, proteins and so forth are released by the microglia and some of the white cells and so forth, signaling, presenting signals to cells and getting them to perform certain functions. turning
49:12
certain genes on and off, and so forth. And that's, that plus the toxins that you get coupled with the inflammatory reaction. Now we get to the last point, which is act on the virus, which is
49:30
latent in the cell, and has in itself its own DNA or RNA, which could release
49:39
a series of programs, reactions that lead to the program development of a cancer cell. Is that correct? Everything we know that can occur with cancer, cancer has to have a stimulation of its
49:54
proliferation. Well, the oncobires, secretive protein, that stimulates AKT, which is the cell signaling apparatus that leads to proliferation. We know that it has to paralyze immune system.
50:11
that virus stimulates STAT3, which also parallels the immune system. It has an artificial protein it produces that is almost exactly like interleukin-10, which is a powerful immune suppress. And
50:26
so when you look at cancer cells, they're microenvironment. There's a cell in there that's a macrophage. It's from the bone marrow that comes into the tumor with the purpose of killing the tumor
50:39
cell Well, the cell surrounding the tumor are secreting agents that suppress immunity,
50:49
mainly inleukin-10, T-regs, transforming growth factor beta, and they all suppress
50:57
the immune system of the cancer cell. So now the immune system can't work, and these cells keep pouring into them trying to, it converts them to what's called Tams, which are truly associated with
51:09
macropaid.
51:11
but these macrophages are no longer M1, which normally kills cancer cells, it converts them to M2, which stimulates the cancer to grow. Absolutely. And we know that the more cam cells that are in
51:26
a cancer cell, the more aggressive it is. Excellent point. So now we've got the body
51:37
in this homeostasis trying to respond to this. And what's happened is the metabolism of the cell has been changed, it's now a tumor cell. It's the creating agents which essentially turn off the
51:47
ability of the immune cells to attack the cancer cell, it shuts that off. And therefore the cancer cell grows without opposition. And now then it's secrete some factors to grow, blood vessels and
52:01
shuts off other mechanisms in the cold cancer cell now that normally would shut the cell down. because the cell has gone awry and it essentially causes its own cell death and they no longer work. So
52:15
the cell keeps growing and growing and growing. And this is the sequence of events that's occurring, right? And you look at a cytomegal virus, for instance, where does it go to infect the cancer
52:29
stem cell? Where does it go to infect in the cell? The mitochondria. So most cytomegal virus in this latent state is in the mitochondria, in the stem cell. And that activates it, and then it
52:44
affects all these things we see in a typical cancer cell. Now, when they looked at cancer cell mutations in the nucleus, most of the mutations had nothing to do with cancer. They had to do with
52:59
development.
53:01
Actually, they make you think that all these things that are mutated in a cancer cell producing the cancer. Most of them have nothing to do with damage. Excellent point. So now here we go. We've
53:13
got a number of factors lined up. We've got cancer, is a metabolic disease. We've got toxins in the environment that are harmful to the cells. And we just gave you an example of a boxing and
53:27
trauma, which is a clear example, but other metals can damage the cells and other agents can damage the cells and so forth So the similar mechanism, we get an activation of the immune system, we
53:41
get over hyperactivity of the immune system, we get excitotoxicity, which means all these other agents are produced, which then trigger the latent virus and then the latent virus goes to work has
53:56
substances and has proteins, which it can't produce. It's got to remember the viruses as a cell to reproduce itself But virus is not life. It's only got DNA and RNA. So it has to live off the cell,
54:10
right? It's a parasite. Right, and what they found about site of megabars is in fact, it has retarded reproduction of the virus. It's mainly producing proteins that produce cancer. Like I said,
54:23
it produces over 100, 200 different proteins. You take one of those proteins and inject it in a mouse, they develop a tumor. Interesting, that's a really important point Now, another question
54:37
here is do we have to have a virus in every cell in the body that's activated, or let's say it's in my thumb or whatever it
54:47
is, or is it the stem cells that are distributed throughout the body, that are the targets in which these viruses act and therefore they change those cells? Yeah, in fact, like if it mostly
55:03
infects stem cell, not somatic cell. And what they found, even with the somatic cell, if it's infected by a cytomegal virus, it reduces the stemness of that cell. It makes the cell stimuli. So
55:18
then it starts acting like a stem cell. Okay. And what they found is when the tascis is, if you take the cancer cell that's not a stem cell, and you implant it anywhere else involved, it won't
55:31
grow. If you take stem cell and let them go to other parts of the body, they produce metastasis. Now, every one of the things that we've just discussed is backed by a research paper that you have.
55:45
And you had the paper and viral oncogenesis, and there's some 100, almost 200 references in there. All of these references are references which support this series of events. And you're working on
55:59
a larger paper on the oncogenesis and virus and tumors. So, attached to all three of these videos that preceded this end of this video, are references where you can find all these things described.
56:17
All of these things have been destroyed. Okay, let me see something else. Now, so we've got it in stem cells. We've got the stem cells throughout the body. We've got the immune cell system with
56:29
that's suppressed. That's another signaling agent that goes out, suppresses the immune cell You told us about that in the macrophages. And now
56:40
comes medicine and says, what I need to do is give this patient cancer chemotherapy.
56:48
And it turns out that both radiation and chemotherapy are toxins. You want to tell us a little bit about that. So we're giving them two reasons. Now, if you take a chemotherapy agent They're the
57:02
confused and the most patient. and you put it in a normal person, there's a high incidence of cancer being induced. One study, they took two cancer droves. They gave it to patients at cancer and
57:15
they developed a second and a third cancer, about 30 of them. So what they don't like to talk about is secondary and tertiary cancers produced by the treatment. We know all chemotherapy is
57:29
inflammatory. All cancers are based on inflammation And if we look at all of these agents, radiation, chemotherapy, they're all inflammatory. And they produce cancers that are much more
57:42
aggressive if they don't cure them, if they don't get rid of everything. And what it is is that it's a balance to try to kill the cancer cell before you kill the patient. And if you don't succeed,
57:55
the cancer comes back, it's much more aggressive. Okay So now, uh, there it is. So we now got chemotherapeutic agents, which are toxic. We're giving toxic and toxic agents to people whose
58:13
cancer has basically been initiated or promoted by or supported by
58:22
toxic agents. And
58:24
we're giving them
58:26
that. But you said that there are other substances that can quell these metabolic reactions, all these reothers, there's millions and millions of every minute of metabolic reactions going on in the
58:38
37 trillion cells. They're just not sitting there. And during the time they're making these things in this body that's trying to achieve almost homeostasis, some of those reactions are going wrong
58:51
and they produce a reactive oxygen, a reactive species and normally the cell immediately quell center attacks it But you said. that also there are some
59:07
supplements, antioxidants that can be used to suppress this overreaction that the body gets into that produces a cancer. Is that correct? Well, yeah, if you look at these things, they're doing
59:23
multiple things that help correct it. It's not just antioxidant, it's also anti-inflammatory, anti-cancer, anti-viral, anti-fungal And they're far safer. And so they started looking at cancers
59:38
and other disease. They found out, well, if you use these, they're accomplishing a great deal in curing the disease. For instance, they looked at antiviral use in cancer. And they found a
59:47
number of cancers
59:52
were far easier to control if you use antivirals
59:59
many of the normal plalenoids, like balkylin, and.
1:00:04
Herculean and quercida are anti-viral and they're particularly directed at the latent viruses like cytomegal virus and the herpesviral. Like asperidin particularly inhibits the herpesviral, which is
1:00:21
associated with a number of cancer. So now we're getting into another problem and that is why don't we know about this? That's why we don't really know,
1:00:35
you can understand it to some degree. We got the wrong idea that cancer is caused by genetic changes in the nucleus and we're going to come to that in just a second. But that's not true and we've
1:00:49
got it as a metabolic disease and we've got a whole bunch of toxins that cause this and
1:00:55
we've activated the body's inflammatory system which makes and hypersensitive work. We're eating in our diet, all this glutamate, which only drives the body more crazy because it's a very
1:01:07
inflammatory agent. And then what we have is a latent virus that's in the stem cells and it's activated. And this goes ahead and produces a whole kind of changes in the tumor microenvironment and
1:01:22
also from this now becoming tumor cell that essentially allow this tumor to grow unadstruct it, right? And now we give it chemotherapeutic agents, which don't work and are toxic. And
1:01:38
what we have an answer for this, which is, well, why don't we work in agents that are anti-inflammatory antioxidants, which essentially quell this whole system down one? And why don't we know
1:01:49
about this, your answer? Well, most of it's in the medical literature. There is a massive amount of information about this The thing is, they don't read it.
1:02:01
They don't study it. They're not taught medical school. They're not taught in the residency, but it's in the literature, a massive amount of it. If you read it and you put it together, you think,
1:02:12
well, this is so obvious, why is this being covered up? Well, being covered up because the pharmaceutical companies are making a mass of money selling these toxic agents that are actually
1:02:23
stimulating cancer growth. We put longing in cancer growth
1:02:30
And we're teaching it in the medical schools and so forth and so forth and so on. So now we have an answer to that. So I'm here in the average person. I'm stimulated by this talk. I'm sitting here,
1:02:43
what the heck do I do? How do I get this? Where do I get this information? There are some sources out there. You write a newsletter about this. We've got the Independent Medical Alliance, which
1:02:58
is beginning to talk about all these things, exactly what you've said. And they published things on this. We'll have these references at the end. So it looks like we're beginning to creep in with
1:03:09
this knowledge. Now a couple of things. Some of these are gonna say, well, wait a minute, Russell. I've had cancer in my family. I've got
1:03:18
generations after generations, they have breast cancer is another example, right? So it must be a genetic cause.
1:03:27
It's sort of a subset of we take this universe of cancers, is there a subset that has a genetic defect or is that genetic defect just accelerating this whole pathway that you're talking about? Well,
1:03:41
it's really an ankle modulation because what happens is they're inheriting the weakness to get cancer. They may never get it. If they're exposed to this pesticide or beside Dr. O'kemko UV light,
1:03:56
they're at a much higher risk of developing it than a normal person. normal genetic and normal mitochondrial function and normal diet. So if you look at people who are born with the BRCA1, BRCA2
1:04:13
genetic defect, which is a repair in line, has to do with a pair of the DNA, well, they have difficulty repairing the DNA. So if the other things we're talking about dietary things, metabolic
1:04:27
thing, allow this cancer to develop at a much higher rate than normally. And that's what's happened is they're in a category of a high rate, for instance, the DNA defect, that causes you to have
1:04:41
an impaired immune system. And then you're exposed to these things. Your late viruses are more likely to be activated and you're more likely to go through the cancer transformation. But it's not a
1:04:55
direct cause of genetic agent It's the fact that your protection against cancer. has been impaired by this genetic feedback. But if we look at the things we know that are genetically related,
1:05:08
they're extremely rare. We're not talking about all cancers, the majority of cancer, they're very rare risk back. Okay, so I think we
1:05:22
just about put the whole picture together
1:05:26
of how we get to cancer, it's not what we thought it was, it's a metabolic disease, a metabolism, we get injuries to the cell, things that promote inflammation, we get the body's reaction to it,
1:05:43
we get a body's overreaction to it, we get the activation of the latent virus in the stem cells, and we get this whole series of
1:05:55
messenger proteins that go out and either shut off the immune system. or turn off the natural killer cells, suicide genes in the cell, essentially allowing the cancer to proliferate and develop
1:06:12
blood vessels by vasogenic factors and so forth. So that's what we've done. We found that if we used, if we thought about this and try to shut this off, it would seem to be the place to shut this
1:06:28
off is that the stem cell, is that right? Or is it at the stem cell and where the peripheral metastasis
1:06:38
is both? Well, once it becomes a cancer, becomes a cancer stem cell, then if we look at all the chemotherapy and radiation, it has no effect on the cancer stem cell. You see, and that's why
1:06:49
cancer reoccur because you did nothing to kill the cancer stem cell. And what we're doing is saying, Well, let's kill what makes the cancer shim shell.
1:07:01
oncogenic virus. The virus that is latent, that has been activated, transforms the cancer stem cell into a cancer stem cell.
1:07:11
And then that produces progenitor cell that make up the bulk of a tumor.
1:07:17
So is the, if we're, we're doing, they're doing this like Einstein did is, from what
1:07:24
you said, as you were sitting down in theoretical physics, you sat down and was thinking about how all these things could happen and come out with this, you've done similar things, you explored
1:07:34
the literature essentially substantiated all the points that lead to the hypotheses you put together with reference materials and actual experiments that have been done not for a year, but for years,
1:07:46
because you started this 20 years, 25 years ago. And that leads you to basically what you're saying. So what's the ideal way to cure cancer?
1:07:59
how dear way is to change your metabolism. And
1:08:04
one of the really brilliant ideas was shown that if you change the metabolism along with hyperbaric oxygen, you get even better effect. It's a tremendous effect because cancer cells, if they're
1:08:20
hypoxic, are infinitely more aggressive. Metastases that are hypoxic are instantly more aggressive
1:08:29
Why would you give them hyperbaric oxygen? Because then you're getting rid of the hypoxia, and what
1:08:35
is driving the cancer is hypoxia-inducible factor one, which is a factor that changes the
1:08:44
metabolin. Brilliant. Brilliant.
1:08:47
So that's a treatment and getting into the anti-oxidants, some of the things that quell all these free radical formations and so forth. That's another So it's a totally new concept in how we're
1:08:58
gonna treat this disease, correct? Yeah, and they're looking at, well, free radicals. Well, they always think of free radicals 'cause they're genetic damage and
1:09:11
that leads to cancer. Well, the free radicals are primarily damaging the mitochondria and they're damaging the cell membrane. I forgot one thing too, and that is, the other thing is the fetal
1:09:23
ketones. Tell the audience about what is ketones Well, ketones are a special-patic product which a cancer cell generally cannot use or energy or anything, you can't use the ketone. So we switch
1:09:40
people to a ketogenic diet generally, which fits out a lot of those sugar, the glucose, and you can reduce the glutamine, but you can't stop it.
1:09:52
And that is cured a lot of cancers, a lot of stage four can.
1:09:59
by this method but if you have to use the ketones to replace the glucose then you don't lose weight you don't have the problems with trying to reduce it now it was thought that you had to go on a
1:10:12
ketogenic diet then you found out no the ketones directly will do it also so you reduce your your intake of these glucose including me you may do intermittent fasting plus the ketones and then that
1:10:31
would do that changes your metabolism keeps the other cells normal and the cancer cells can you love but you are right you'll survive because you can metabolize it the yeah the normal cell can
1:10:45
metabolize the ketone but the cancer cell can't therefore you basically starve it to death and then you give all these other antioxidant-free radical factors. And that might be the direction we ought
1:10:59
to go. Is that correct? Sure, I mean, it's a multi-factorial attack. You're doing everything that harms only the cancer cell. When we dealt with infection, we talked about the magic bullet that
1:11:15
would go to the infectious agent and kill it, but not harm the normal cell. Well, we need a magic bullet for cancer also. If you look at chemotherapy, chemotherapy harms also Normal and cancer.
1:11:28
To look at radiation, hammer, and harm the all cell. Normal and cancer, sir. But now we want to do it with the magic bullet where the normal cell can metabolize its ketone, but a cancer can.
1:11:43
Excellent. So is there anything, I think we've got a very natural series of events here that we started with,
1:11:52
Cancer is a metabolic disease. It's not a cause by a whole bunch of genetic changes. It's a metabolic disease. It's a disease which then is
1:12:06
stimulated by a whole bunch of variety of different toxins which cause inflammation, the activation of the immune system with an overreaction and excitotoxicity which releases a whole bunch of
1:12:21
transcription factors which then directs the cell in a different direction and activates the latent virus which then is in particularly in the stem cells. The stem cells then convert their metabolism
1:12:36
to the Warburg metabolism where they sidetrack the glucose from making all the energy and it goes to
1:12:47
pyruvate and lactate and then it takes over the metabolism of the changed cell and the virus end. has some genetic either RNA or DNA, probably there's a sequence of
1:12:58
proteins and coated in
1:13:01
it, which are released, which essentially suppress the activation of the immune system. Stims shut off the killing mechanism, the cell, have formed other kinds of proteins that make vascular
1:13:13
growth possible, metastasis possible, and chemotherapeutic agents in radiation are harmful, and do the same thing the toxins do. And so either you put them on a ketogenic diet, which essentially
1:13:28
stars the cancer cell, supplement it with antioxidant, anti-inflammatory agents. So you're, I think that's about a two minute summary of what we talked about, but is that correct? Yeah, it's
1:13:43
all correct, all correct. Anything we missed? No, I mean, just people keep in mind
1:13:55
that the glutamate is extremely harmful and your diet is pro-inflammatory as you get older, older people get more inflamed than younger people and these carcinogenic viruses tend to go into senescent
1:14:07
cells, older cells. And they tend to make younger cells into senescent cells. Senescent cells secrete inflammatory chemicals that are very powerful So the whole thing is being driven by
1:14:20
inflammation. And if you look at diabetes, for instance, cancer rates and diabetics are far higher than people with normal blood sugar control. Why? Because diabetes is inflammatory. Why do they
1:14:34
get blind? Because it's inflammatory. Why do they have amputations? Because it's inflammatory. So it all goes back to your metabolism It is pro-inflammatory and it's driving the activation of all
1:14:50
these viruses. which drives the cancer.
1:14:54
Rossignol, outstanding, outstanding interview, outstanding, made this all make a lot of sense. I hope for our listener and
1:15:05
just a terrific, terrific job. Okay. Well, thank you, Jim, you have some great questions. Okay, terrific. Well, we just hope everybody learned something that is, we just are trying to give
1:15:17
you the truth that, so it's different than what you know It's different when people have told you, but there's abundant evidence, abundant evidence for every statement
1:15:30
that Russell has made it, and he spent years putting this all together into a logical sequence, and along the way, people have proved what he said is true, and then that's how it's happened.
1:15:41
Right? Wrong. All right. I'm gonna close this down now, just a minute here key references and charts. For Dr. Playlox presentation today, be prepared to take screenshots so you can have this
1:15:57
information for your records. This is one of the slides we showed previously on his background, on the wellness newsletter, and on the subjects of the newsletter, two of them were on cancer, is
1:16:15
actually a metabolic disease and natural compounds offer more benefits for cancer treatment. In a paper written in Surgical Neurology International in 2019, which comprises a great deal of the
1:16:31
subject of this presentation, it's accelerated cancer aggressiveness by viral-onco-modulation.
1:16:39
Six years ago, he wrote, New targets and newer natural treatments for cancer control and treatment.
1:16:47
These are series of of 11 references. These are the first five. Take a screenshot of these, and it relates to the subjects you presented in the talk.
1:16:59
This is the second sort of references.
1:17:04
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1:17:15
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