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SI Digital, Innovations in Learning, the 3D Live video journal, Interactive with Discussion. Now offering this program and others on podcasts on Apple, Amazon, and Spotify are pleased to present
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an association with SI Surgical Neurology International, a 2D Internet Journal
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Another in the series of Dr. Blalock reports on medically related controversies today.
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The topic of this program by Dr. Blalock is atherosclerosis, cholesterol, statins, complications, immuno-exciter toxicity, and nutrients.
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Dr. Blalock is a head of theoretical neuroscience research and an associate editor-in-chief in the neuro-inflammation section. in surgical neurology, international, and SNI digital.
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He's a board certified clinical nutritionist, creator of the Blalock Wellness Report and author of multiple book scientific papers and a health commentator on radio TV in and the epic times. These
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are some of his books on natural solutions in healthcare in regard to liver cure, natural strategies for cancer patients, natural health, excitotoxins, which is a topic of today's talk and was
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co-authored with Dr. Auslin on the bookThe China Virus, What is the Truth? He also publishes a monthly nutritional newsletter entitledThe Blalock Wellness Report. You can subscribe at
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wellnessreportnewsmaxcom. What's this pick today?
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one of the two most controversial topics we have, which is, quote, hardening of the arteries, high cholesterol, which everybody believes related to that, he's gonna talk to you about that, and
2:12
what you can do about it. Russell, you wanna take it from here? Yeah, I think that your introduction is right on target, and basically this has been a fascination with me for a long time, for
2:28
decades. And I looked at the cholesterol theory, and I went to a number of lectures, and I listened to some experts, and I read their papers, and I said, Well, there's a defect here. There's
2:41
something wrong that we have a drugthat supposedly is lowering the cholesterol, even lower than normal, and heart attack and strokes for the number one death killer in the world. And supposedly
3:00
these statin drugs, lowering cholesterol is gonna solve the problem. But death rate from heart attacks and strokes is still the number one killer.
3:11
Our rate hadn't fallen virtually any. So what I did is I researched the history of how cholesterol ever got chosen as the cause of atherosclerotic plaques
3:25
And what I found was that you could inject cholesterol in an animal, no matter how much cholesterol you injected, you could not produce a plaque. You had to add omega-6 oils to make it do anything.
3:37
And
3:43
recently what the researchers have found is that without an elevation of cholesterol, but you increase the amount of omega-6 fats you get plaques. And all the research in acid scrosses now says,
3:57
well, acid scrosses is not due to high left role, is due to inflammation. And the inner lining of the artery, the endothelium becomes inflamed. And that inflammation begins very early in life.
4:12
They even find it in some small children. But the real difference of what we found about plaque occurred during the Korean War, when you had young men die, that were in excellent physical condition,
4:29
and all of them had fatty streaks in the arteries. And so they said, well, you know, this is kind of strange that these people that are in excellent condition have these fatty streaks so early in
4:43
life, and then they're dying when they reach 55 to 65 years old, they're having a stroke And this is a. process of these fatty streaks. Well, most people, including physicians, believe that the
5:03
cholesterol accumulation as a plaque produces physical obstruction of a major vessel in the heart or gutted order. When in fact, oftentimes, it's a smaller plaque that produces the heart attacker
5:19
stroke and does not include the vessel by the accumulation of cholesterol with the fact that the plaque ruptures. And when it ruptures, it releases polyinflammatory fats, which trigger a thrombosis
5:34
and the thrombosis is sudden. And that's why a heart attack is so sudden.
5:40
So it results indicated that the idea that the atherosclerosis was slowly occluding the vessel was not true. And so we had two things to consider. One,
5:54
What was the contribution of the fat? Where does it come from? What was causing it to accumulate in the walls of the vessel? And we found that hypertension, where the arteries bifurcate, there's
6:09
terrors or damage to the endothelium, and the fat accumulates in these areas. Well, the fat accumulates in the areas, and it's inflammatory So the body has an immune response to it. The
6:25
macrophages are attracted to the fatty deposits, the fatty streaks, which cause no symptom.
6:34
And once the macrophage enters this, it starts engulfing the fat particles. And it produces a highly inflammatory macrophage cell. And we call those foam cells
6:47
The immune attack continues, so it's purely inflammatory at that point. or mainly inflammatory at that point. And so the body reacts to try to encase this plaque and it produces a fibrous cap over
7:04
the plaque to keep it from rupturing because as long as it's contained within the wall of the vessel, the blood can flow past it.
7:15
And what we found to our surprise was that it wasn't the largest plaque that was producing most heart attacks and strokes. It's oftentimes a slower plaque and that plaque had ruptured. The larger
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plaque had not ruptured.
7:32
And so it changed kind of the thinking we had of the whole process. The
7:39
reason that cholesterol was chosen was because of pharmaceutical companies figured a way to lower cholesterol because it usually I'm not about it pathway,
7:52
Coenzyme Q-10 also uses and you could lower cholesterol, but you couldn't lower the fat, that was the number one fat found in the plaque, which was in the omega-6 fat, not cholesterol. So can I
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just ask you a question to me? It says it's a superb summary. But you said
8:13
they were working on cholesterol and I missed a segment where you said, what was interfering, what were they doing? They interfered with absorption of the cholesterol and so forth, and it's not
8:27
important anyway, because it doesn't produce the plaque. But I missed something in there. Well, there was a pathway for the production of cholesterol and the pathway produces two essential
8:40
elements, one cholesterol and one coenzyme Q10.
8:44
And cholesterol, of course, is used extensively by the brain It protects the cells against carcinogenic changes. it produces steroid hormones, it has a lot of functions.
8:56
And then they found that if you block this pathway, you're blocking also CoQ10, which is used by the brain, which is used by all cells to produce energy. And you're also reducing the cholesterol
9:11
that is used for all these other important functions. So a number of people, a number of people who've had this cholesterol severely lowered develop amnesia, they develop dementia, they develop a
9:26
number of neurological conditions, peripheral nerve problems, and they have heart problems and increased cancer rates. And it starts to make sense that you're interfering with a formation of a
9:42
critical chemical cholesterol. And cholesterol is not the number one cause of the plaque The plaque is caused by inflammation. of the fatty streak
9:54
and accumulation of oxidized oils. So what they found as they began to look at cholesterol, they were struck by the fact cholesterol being high, which we know it metabolically, some people can
10:09
have enormously high cholesterol and no atherosclerosis. And so they said, well, we gotta find an answer for that. And so what their answer was, well, there's some fat called cholesterol that is
10:22
oxidized. And it's only the oxidized cholesterol that can produce atherosclerosis. Unoxidized cholesterol has nothing to do with it.
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And then later, then this is recently, as researchers looked at, well, it's not cholesterol, it's omega-6 fats and they're polyunsaturated and very easy to oxidize often they're oxidized before
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you eat them.
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Most people have not looked at, well, where's their oxidized omega-6 coming from? So a study of restaurants found, they're using the oils over and over and over for months. And by the time people
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are eating the foods cooked in these oils, they're already oxidized. They're fully oxidized from the heat And then when they end up in the blood, they're already oxidized and they're producing
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esclerosis.
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So what you're talking about is a fundamental biochemical mechanism. And if you try to block the absorption of cholesterol as the pharmaceutical companies are working on, not only do you block the
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absorption of cholesterol, which you need for many cell membranes throughout the body and other kinds of things that are very important. But you're blocking CoQ10.
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which is a metabolic cofactor in a variety of other metabolic reactions. And without that, you're also subjecting yourself to another set of metabolic problems, is that right? That's right. And
12:03
some of them are serious, for instance, Parkinson's disease. We know that the number one defect is the complex one, which is reversed somewhat by CoQ10
12:16
So if you don't make CoQ10 and your CoQ10 is deficient, then that's interfering with complex one. So if we have it, what you're saying is we fit animals, high doses of cholesterol, it didn't
12:31
produce plaque. Then what happens is the pharmaceutical companies say, well, I can find a way to lower cholesterol, but they developed a drug that not only lower cholesterol, but you need it and
12:45
it prevented you from getting CoQ10. So it wasn't helpful, and it was harmful, and they didn't tell anybody. Well, in the beginning, they used to add co-Q10 to the statins, and then they came
13:00
to the conclusion, that's gonna scare people, so let's just leave the co-Q10 out. But your muscles also, your striated muscles, and your heart's a striated muscle as well, use an enormous amount
13:15
of co-Q10 Well, what's the number one complication instead? Weakness, destruction of muscles, pain. And we found the cure for that is how dose is a co-Q10.
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And they don't mention that, that's not mentioned anymore, they advertise, lower is better. And they did, and one of their primary
13:41
studies, which used a lot of people found, in fact, if you lord it ah there was a significant increase in death from intracranial hemorrhage. That was never mentioned in the conclusion or the
13:56
abstract. What caused that? You had to look at the data. And the data showed the number one cause of death was from the statins was brain hemorrhage.
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Why did the hemorrhage occur?
14:12
Well, it's never been research Nobody really, they tried to hide it out, hide it completely from the positions in the public, the hemorrhage. And so they kept, they kept advertising, lower is
14:27
better. Well, I was at a lecture one time and I showed an advertisement for one of the major statins. And I said, I blew up little tiny writing that was in the corner. I blew it out and I said,
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read this, that there was a 30 drop in death rate
14:47
And I said, the statisticians took this and looked at it, one of the top statisticians in the world. And he said, that's funny. The real decrease was 1, 99 it had no effect. And what they were
15:05
taking was a percentage change of a percentage, not the real reduction. And so I tell patients, I said, well, are you willing to face these serious complications, knowing 99 of the time the drug
15:22
is not gonna do you any good at all.
15:25
And that becomes a major thing. Well, you're constantly hearing, oh, these are rare, these are rare. And I said, well, if it occurs in you, the incidence is 100 of the percent made. And so
15:39
if you live with it or you die with it. So to tell a patient, well, Oh, oh, that's rare.
15:47
That doesn't tell me anything. If it happens to me, it's 100. Absolutely right. Eventually what they looked at is, there's a difference in the kind of cholesterol. You see, we heard all these
16:00
things about LDL cholesterol, it's dangerous, it's the bad cholesterol, and HDL is the good cholesterol. And the researchers were looking at it and said, Wait a minute, that's not true at all.
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There's two forms of LDL cholesterol And one is the small dent, which is easily oxidized, and then there's a large buoyant, which is not easily oxidized, and in fact, it's beneficial. And so
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it's not really bad versus good, it's beneficial versus harmful. Well, there were two forms of LDL. Well, if you go to a doctor and you get a blood test, he'd tell you, Blick, your LDL is high.
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If you don't have a differential, how do I know that most of it is large bullion? Well, it's good. And what they found is that large bullion LDL is as helpful as the HDL cholesterol. And then
17:02
they found, well, there's a difference in the HDL cholesterol. The HDL cholesterol contains a group of enzymes that actually act as an immune cell, an immune function. And that when you get
17:16
infected, your HDL goes up, infection goes down, the HDL goes down. And that HDL, like LDL, there's good ones and there's bad ones. Harmful and unharmful. And so you can have a high HDL, but
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you have a defect in the enzymes inside of the HDL, and now it's harmful. So your doctor says, Oh, that's great you have a high HCL. Well, it's not necessarily great. If you're inflamed,
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actually it's harmful. It's as harmful as a small dense LDL cholesterol. So the basic story here is we needed differentiation of the different types that not all LDL is bad and not all HDL is good.
18:06
And that's been bared out. Numerous researchers have looked at this and said that's exactly true Well, if you're an inflamed person, let's say you have rheumatoid arthritis or you have autoimmune
18:17
disease or you're getting older and you're becoming more inflamed, then the different forms of LDL either are gonna help you or they're gonna harm you. And so if you have a lot of small dense LDL,
18:34
that inflammation is gonna inflame the cholesterol and oxidize it, then it could be harmful. but it's also oxidizing your omega-6 fats. And what most people don't know is while omega-6 is an
18:51
essential fat, it's been determined that the Western diet just has about 50 times the amount of omega-6 that you need. And that their omega-6 is very dangerous. It's linked to Alzheimer's disease,
19:07
Parkinson's disease, all neurodegeneration, Parkinson's disease, atherosclerosis, blacks, elevation of blood pressure. So a number of things are connected to this type of fat, their omega-6 fat.
19:25
And it's not because your cholesterol is high, and they never look at the fact that, well, you have tens of millions of people on statin drugs, and yet it's still the number one killer Well, it
19:39
should have disappeared. We shouldn't have. any of those tens of millions of people dying of heart attack or having heart attacks. But if you look at the major studies that they like to quote,
19:52
that's exactly what you're seeing. Well, your statin drug was no better than aspirin. And then you look at drugs that lower cholesterol but have nothing to do with the mechanism of statins, there
20:09
was no effect, no benefit
20:12
And so then we started, what could be the small benefit that 1 I'm talking about? Well, statins are an immune suppressant
20:22
and they suppress your inflammation by suppressing your immunity. Well, that puts you at risk of cancer, that puts you at risk of infection, that it puts you at risk of COVID,
20:35
a number of things.
20:38
And that's not even considered that you've taken this patient And in fact, the transplant. surgeon said, we found that the statin drugs prevent rejection of the organ because they're suppressing
20:53
immunity. That's how powerful they are.
20:58
So these are the considerations that you need to make. And of course, the brain makes its own cholesterol, which is also inhibited by this statin drugs And if you don't have that cholesterol, your
21:13
brain function is
21:17
affected. And that's why people have seen numerous cases of amnesia, forgetfulness, and loss of memory that are on statin drugs, because it's interfering with the cholesterol function in the brain.
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OK, so what we've got here - This is terrific. we got here is the fundamental observation that if you're given animal intravenously a large amount of cholesterol, it does not produce a plaque.
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Okay, then we get to the point is, okay, that didn't work. So we got to figure out what some of the plaque is because we take the blood vessel and we look at it, it looks like there's fat in the
22:01
plaque. And so people have kind of then tried to differentiate into high and low density cholesterol. I mean, I just had some chemical tests done and it had high and low. There was no
22:15
differentiation, as you said, into the good forms of high and low and the good forms of low. And so it's meaningless. So now the cholesterol level I get and the blood test is meaningless. The HDL
22:29
and the LDL level is meaningless. But you said something and that was that people in our culture and American culture have 50 times the high, 50 times the level of omega-6 fats in their blood as
22:46
they do in others. Well, that would seem to be one of the correlates here. So it's basically what we're eating. Is it, am I right? That's right, 'cause a lot of these omega-6 are oxidized
22:60
before you eat 'em. For instance, almost all salad dressings are omega-6 oils, peanut, safflower, corn, coconut oil, all these oils are omega-6 oil. And so, once they're out of the bottle,
23:14
once it's unsealed and exposed to the air, it oxidized. So you're eating oxidized oil. And it's beyond dispute that oxidized oils when consumed reduce atherosclerosis. Oh, okay, well, what does
23:31
the oxidase, the audience is going to know, what does the oxidation do to the molecule? Why is that make it so dangerous?
23:42
Well, a polyunsaturated fat has numerous unsaturated bonds. And so they're easily oxidized. You never cook with a polyunsaturated oil because it oxidizes it. You don't expose it to air because it
23:55
oxidizes it. Well, when you consume an oxidized oil, it produces inflammation. The inflammation produces free radical generation and lipid peroxidation. Well, what's the number one cause of
24:10
atherosclerosis? Heart disease and neurodegeneration. Free radicals, lipid peroxidation. Exactly what we're seeing by the consumption of these enormous amounts of polyunsaturated fats. Well, you
24:26
remember that they used to advertise all the time that corn oil lowered your cholesterol and was such a good oil. And doctors were telling patients eat more corn or all our peanut oil. called that
24:42
lauria cholesterol. They were doing just the opposite of what should be done. They were increased in the intake of oxidized oil. And that's how people ended up with this problem. And you look at
24:56
restaurants, well, the cheapest oil, the most common use oil is peanut oil. And peanut oil has the closest correlation to atherosclerosis and heart attacks. It's producing the inflammation and
25:09
the lining of the artery and producing the fat accumulation, fat oxidation, and the bone cells, which are the macrophage trying to engulf this inflammatory fat. So we have in our culture then,
25:23
because of the habits we've evolved. I'm sure we're going to get into this and some of the things we eat, which are because we all evolved from really a fist-based culture, which has a low omega-6.
25:37
I tell me if I'm wrong. And as opposed to a higher Omega-3, and the Omega-6 then gets oxidized with all the things that we do to it, cooking it and so forth, the goods exposed to the air, and
25:53
that then becomes a pro-inflammatory agent which will lead to the formation of a plaque in the process of inflammation because certain cells come in and they absorb all this and they perform a plaque.
26:07
Is that correct? That's exactly right So now the audience is saying okay, I just learned cholesterol isn't the issue, I learned that high in low density cholesterol is
26:20
more complicated than I thought but my doctor doesn't tell me about it and now I know I've heard about this about omega-6 and omega-3 fats and what substance was animal has a high amount of omega-3
26:38
fats and what so is that
26:41
Well, usually it's a small fish. What happened is the large fish eats a smaller fish. The smaller fish has the highest content of omega-3 fat. That would be sardines or something like that. Right.
26:54
And salmon. And salmon. And the large fish is consuming these smaller ones, and that's how they get to DHA content. Well, if we look at DHA, it almost erases beta amyloid in the brain. Now DHA,
27:13
what is the DHA, is that omega-3? It's
27:17
a component of omega-3, or omega-3 are made of EPA and DHA. Oh, okay. It's also a polyunsaturated fat cook with it.
27:29
But we found that if you feed it to animals with high amyloid, the amyloids erase in the brain and a hyperphospholated tau protein is the right.
27:42
it returns to normal. It's an anti-inflammatory oil. And we found that heart attacks and strokes are really less if you usually omega-3 over to omega-6. And that omega-6 will displace the omega-3s
27:59
in your body. So in fact that we changed our diet, we don't eat grass-fed beef, which actually has some omega-3 in it We eat mainly beef that has spread grains, and the grains have a high omega-6
28:14
in it. Okay, that's great. So it's gonna, we're gonna find it in meats. We're gonna find a high omega-3 in fish. It can be in salmon, it can be in sardines, and so forth. So now we know what
28:29
dietary situations we should be in. And we know a little bit about if we're ingesting a lot of, well, if we're ingesting the meat. What is it in the meat that's got the high omega-6?
28:47
Well, the cow will normally have some omega-6 in and it's from his diet or the diet of the cow or the cow. Okay.
28:58
And it's because they're not being fed grass, they're fed grains, grains are high and omega-6 fat. Okay, some people say if you have fatty meat that that makes the difference, is that true? You
29:13
know? Well, if you look at the literature, there's almost no connection consumption fat saturated between
29:22
and these diseases. Well, that makes sense 'cause saturated fats are not so low, they're not inflammatory. But an overabundance of saturated fats can interfere with insulin function.
29:35
And that would be the only connection. If you look at these people that have high cholesterol. Most of them have metabolic syndrome. And it's because they're consuming so many carbohydrates and
29:51
simple sugars, it raises the bad LDL, and then they get a metabolic syndrome. Well, a metabolic syndrome is a combination of hypertension, type 2 diabetes, and an elevated lipid profile,
30:07
particularly these inflamed fats.
30:12
And so when they look at it and they blame it on cholesterol, almost everyone of these patients is a metabolic syndrome. They're not pure elevated cholesterol. When you look at a genetic elevation
30:24
of the cholesterol, there's no increase in heart attack and stress. So the take-home message here in this part, for the person who's listening to this is, Look, I expect my doctor to tell me that
30:39
she's not going to know. He's not going to get a differential. He's going to get the blood test. It won't tell me what I want to know. What the amount of cholesterol this doesn't make a difference.
30:48
And the LDL and the HDL has to be fractionated to be of any value whatsoever. But I can do something myself. And that is I can go out
30:60
and find omega-3 sources, which are basically fish, because it's not the meat, because they're fed green, which has a lot of omega-6. So I can do something about it. I know that my been on
31:16
statins, because my doctor - I'm not talking about myself, but the doctors put me on statins. And I know, because they told me, one of the things is muscle weakness. And you just said earlier
31:27
that if they want to treat it, which the statin does, and they want to block it,
31:34
they're blocking also an essential cofactor, which is coq10, helps your muscles so you're gonna make your muscles weak. And then you just said that your hook is still gonna cause a disturbance in
31:46
brain function. Right, that means goes with the neurodegeneration, but what we call is a rhabdomyolysis. And
31:55
what happens is the statins in a number of cases, about 3 of cases will produce a fatal destruction of the muscles, so much
32:09
inflammatory destruction is released by the muscles, it damages the kidneys, the kidneys fail and the person will die Well.,
32:18
their response is, well, this is a small percentage, but we go back to say, well, if it happens to you, it's 100. And I remember reading the case of a guy with about 35 years old, and he had
32:31
cholesterol elevate He felt just fine, so his doctor put him on set.
32:39
pain, so much weakness, he couldn't get out of bed or work. Yeah. Okay. And so, they did another research to see, well, what happens to the muscle when lower doses of statins, that's
32:51
asymptomatic has happened? Well, they found out there's muscle destruction occurring as well.
32:58
Okay. Now, I'm either a doctor or a melee person. I'm listening to this. I go to the doctor and I say, wait a minute I just heard Dr. Blalak tell me that the statins you've been giving me are
33:11
going to be harmful to me. And then if I ate a diet that's low in omega-3s, I don't need this. What's the doctor going to tell them? Well, most of what he gets is pamphlets from the
33:24
pharmaceutical company anyway. And he goes to a meeting and they tell him the same thing. And the people who put the meeting together are influenced by the pharmaceutical companies So they're just
33:35
hearing one side of the story, they're not listening to the science. And if you go and look at the science articles, look at this, they all say there's no question it's inflammatory, it's not a
33:47
cholesterol problem. And so the doctor really doesn't understand the pathophysiology of atherosclerosis, the history of atherosclerosis, and the fact that if you take an animal that normally eats
34:03
plants and you feed them high cholesterol or cholesterol diet, they develop atherosclerosis. But if you feed them the diet they're used to, they don't develop atherosclerosis. Are there certain
34:15
kinds of animals
34:19
like veal or chicken that have a lower levels of omega-6? Oh yeah, as long as if you eat organic meats, organic meats are fed by nature. So insects that a chicken is eating or roosters eating
34:39
Omega-3 fat, not Omega-6 fat. Interesting, okay. That's the difference.
34:46
Okay, and what about veal, that's a young calf. If veal would be good, as long as it's organic veal. The problem with veal that I found is that mad cow disease, what they were doing at one point
35:02
is so that the inspector would not detect the cow diet of mad cow, they would kill it when it was young. So the veal is more likely to be infected with the prions than if an older cow would have
35:20
catalyzed, would have died. Yeah. Okay, that's a very good practical take home. Now, where are we going with this is, are there any drugs that are gonna make this better or what you're saying
35:25
is just get on a normal diet.
35:39
Well, mainly change your diet. Change of your diet. And what you find is that people are all excited about the fact that you stop eating processed foods and eat organic foods. The incidence of
35:51
heart attack strokes or atherosus just plummets. Just absolutely plummets. And if you take antioxidants, take anti-inflammatory, which is the primary cause of the disease, then that prevents the
36:05
disease
36:08
So the statins, the only reason they work at all is 'cause they're knocking out your immune system. You're having trouble forming an inflammation, but at the cost of the fact, now you don't have a
36:20
functional immune system.
36:22
Is it everybody who's taking statins have a suppressed immune system? Yes, yeah. If you say like I said, the transplant surgeons were saying, let's give them statins because it expresses their
36:36
immunity and they won't reject the Oregon.
36:39
So we have a huge segment of the population that likely is on statins. It's suppressing their immune system. And we're not, I mean, we didn't wanna get into that today, but I think a little bit
36:50
into that. Is that exposed the individual to more types of infectious disease that they are gonna be incapable of counteracting? That's right. In fact, I went to a meeting to speak. I was a
37:04
speaker at a Jamaica meeting and I met one of the researchers who was
37:10
an expert in statins. And she said, you know, statins, consumptions associated with a higher incidence of ALS
37:22
and peripheral neuropathy and a whole bunch of other diseases, some of which are crippling or deadly, like ALS. And she wrote a very thick article about all the complications
37:38
with a statin. Nobody receded that, you know, or
37:43
research in that area was impeccable. Outstanding. So one other thing, Mediterranean diet, it's basically fruits, it's vegetables, it's leafy vegetables
37:58
and fish.
38:04
That and omega 3 oils out of your fish and omega 9 oils from the olive oil. The olive oil contains phytochemicals that prevent the inflammation and also slightly thin the blood.
38:18
I remember back when I was beginning to study this, they were saying
38:34
that omega 3 oils doesn't work. Well, I looked at the placebo, the placebo was olive oil and I said, well, the placebo is doing the same thing as the omega 3 is doing. inactive, which is
38:38
supposed to be a plea to a placebo. And we're seeing that more and more that the placebo has activity. And that way it looks like what's so better than placebo.
38:51
Well, it's just an outstanding explanation. It's going to cause a lot of uproar. In your newsletter, do you cover, I know you had a newsletter about cholesterol, will this be in your newsletter?
39:06
Yeah, actually, I've written about four or five newsletters about what I found about cholesterol. One covered the history. It went back all the way to the 30s when he was looking at the research,
39:18
how it was done, how they concluded that it was the cholesterol. You see, one of the things I found is that they couldn't get the animals to develop atherosclerosis by injecting the cholesterol.
39:33
But when they added omega-6 oils, then they would produce of atherosclerosis. And so they just assumed it was a cholesterol. So when you give me the references for this, would you add the ones
39:46
from your newsletters because they can go back and get the newsletters and look that up? I think that would be terrific. Anything we didn't cover at this point, obviously we didn't go into vessel
39:58
occlusions and so forth and so on.
40:02
Anything about cholesterol and statins we didn't cover that you want to talk about? Well, there was something I became friends with Dr. Lomol who was a cardiovascular surgeon and he's an expert in
40:16
lymphatics and he said one of the things they found out is if you occlude the lymphatics to the heart, it develops enormous acid sclerosis
40:27
and so he hypothesized that there was a defect in the lymphatic drainage of the vessel itself
40:36
accumulation of these fats. And so I thought that was fairly interesting. But he said it was reproducible. Every time they were occluded to lymphatic drainage of these vessels, they produce this
40:50
severe atherosclerosis. The other thing is the endothelium of the vessel. The endothelium is what I call the brains of the vessel. You know, vessels dilate when they need a greater flow of blood
41:06
and they constrict when there's less need of blood, it was adrenaline. So they're reactive. Well, they react through the endothelium
41:17
of the cell, which affects the muscular layer. And there's damage to the endothelium is what produces this in the first place, and it's due to inflammation. And so there are a number of natural
41:34
products, which protected endothelium. And so I emphasize that if people want to protect their arteries, they need to protect their endothelium, the brains of the blood vessels. And for instance,
41:49
nanocircumum, nanocarcidum,
41:51
saffron, these things protect that endothelium.
41:57
Okay, so that's terrific. I think summary of Dr. Blalock's presentation today is that basically an arterial plaques were found in young Korean more deaths, and cholesterol was not the cause of
42:13
those plaques. Intravenous injection in animals fails to cause plaques, but omega fats injections and omega-6 fats injections did cause plaque formation. Pharmaceutical companies promoted statins
42:29
to lower cholesterol, you though, there was no benefit.
42:34
and there were measurements of cholesterol, HCl, and LDL, which were also of no benefit because they were not detailed enough. Statins lower cholesterol and co-cutent, causing muscle weakness,
42:48
memory loss, Parkinson's disease, and suppress the immune system leading to infections in COVID.
42:59
Plaque is basically caused by injury to the vessel wall, inflammatory cells entering the wall at the site of injury, omega-6 fat circulating in the blood, entering the cells and forming foam cells
43:15
with the resulting formation of immuno-excitotoxicity, plaque growth, rupture, blood plant, leading to stroke and myocardial infarction. The treatment is Mediterranean diet, composed of fish,
43:32
Omega-3 oils, antioxidants, vegetables, fruits, anti-inflammatory agents. Others in the Doctor Blaylock Report series, whether the facts
43:45
are programs on the bird flu, monkeypox, dangerous in microwave radiation, is for a dangerous to your health, measles, whether the facts and fiction in regard to that disease, diabetes, causes
44:01
of obesity, and this program's standards, which are the wrong treatments for plaques and have toxicity.
44:12
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44:31
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44:37
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44:43
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44:52
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